Pilocytic astrocytomas do not show most of the genetic changes commonly seen in diffuse astrocytomas

Histopathology

Volumn 37, Issue 5, 2000, Pages 437-444

  Cheng, Y ^a   Pang, J C S ^a   Ng, H K ^a,e   Ding, M ^b   Zhang, S F ^c   Zheng, J ^a   Liu, D G ^d   Poon, W S ^a  

^a CHINESE UNIVERSITY OF HONG KONG   (Hong Kong)

^b ANHUI PROVINCIAL HOSPITAL   (China)

^c SICHUAN UNIVERSITY   (China)

^d BEIJING HOSPITAL   (China)

^e CHINESE UNIVERSITY OF HONG KONG   (Hong Kong)

Author keywords

CDK4; p16; p53; Pilocytic astrocytoma; PTEN

Indexed keywords

PROTEIN; PROTEIN P16; PROTEIN P53;

ADOLESCENT; ARTICLE; ASTROCYTOMA; CHILD; CLINICAL ARTICLE; CONTROLLED STUDY; FEMALE; GENE LOCUS; GENE MUTATION; GENETIC ASSOCIATION; HETEROZYGOSITY LOSS; HUMAN; HUMAN TISSUE; IMMUNOHISTOCHEMISTRY; MALE; MICROSATELLITE INSTABILITY; PHENOTYPE; PRIORITY JOURNAL; SINGLE STRAND CONFORMATION POLYMORPHISM;

ADOLESCENT; ASTROCYTOMA; BRAIN NEOPLASMS; CHILD; CHILD, PRESCHOOL; CYCLIN-DEPENDENT KINASE 4; CYCLIN-DEPENDENT KINASE INHIBITOR P16; CYCLIN-DEPENDENT KINASES; DNA MUTATIONAL ANALYSIS; DNA, NEOPLASM; FEMALE; HUMANS; IMMUNOENZYME TECHNIQUES; INFANT; LOSS OF HETEROZYGOSITY; MALE; MICROSATELLITE REPEATS; MUTATION; PHOSPHORIC MONOESTER HYDROLASES; POLYMORPHISM, SINGLE-STRANDED CONFORMATIONAL; PROTO-ONCOGENE PROTEINS; PTEN PHOSPHOHYDROLASE; TUMOR SUPPRESSOR PROTEIN P53; TUMOR SUPPRESSOR PROTEINS;

EID: 0033731127     PISSN: 03090167     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1365-2559.2000.01005.x     Document Type: Article

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