The ability of HIV type 1 to use CCR-3 as a coreceptor is controlled by envelope V1/V2 sequences acting in conjunction with a CCR-5 tropic V3 loop

Proceedings of the National Academy of Sciences of the United States of America

Volumn 95, Issue 13, 1998, Pages 7682-7686

  Ross, Ted M ^a   Cullen, Bryan R ^a  

^a DUKE UNIVERSITY MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CD4 ANTIGEN; RECEPTOR SUBTYPE; VIRUS ENVELOPE PROTEIN;

ARTICLE; BINDING SITE; CHIMERA; HUMAN IMMUNODEFICIENCY VIRUS 1; MOLECULAR CLONING; NONHUMAN; PRIORITY JOURNAL; RECEPTOR BINDING; RECEPTOR UPREGULATION;

ANIMALS; CELL FUSION; CLONING, MOLECULAR; COS CELLS; HIV ENVELOPE PROTEIN GP120; HIV-1; PEPTIDE FRAGMENTS; RECEPTORS, CCR5; RECEPTORS, CHEMOKINE; RECEPTORS, HIV; RECOMBINANT FUSION PROTEINS; STRUCTURE-ACTIVITY RELATIONSHIP;

HUMAN IMMUNODEFICIENCY VIRUS; HUMAN IMMUNODEFICIENCY VIRUS 1;

EID: 0032560559     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.95.13.7682     Document Type: Article

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