Basic FGF attenuates amyloid β-peptide-induced oxidative stress, mitochondrial dysfunction, and impairment of Na+/K+-ATPase activity in hippocampal neurons

Brain Research

Volumn 756, Issue 1-2, 1997, Pages 205-214

  Mark, Robert J ^a   Keller, Jeffrey N ^a,b   Kruman, Inna ^a   Mattson, Mark P ^a,b  

^a UNIVERSITY OF KENTUCKY   (United States)

^b UNIVERSITY OF KENTUCKY   (United States)

Author keywords

4 Hydroxynonenal; Alzheimer's disease; Apoptosis; Diethyldithiocarbamate; Dihydrorhodamine; Hippocampal neuron; Lipid peroxidation; Mitochondrial transmembrane potential; Neurotrophic factor; Ouabain; Superoxide anion radical

Indexed keywords

4 HYDROXYNONENAL; ADENOSINE TRIPHOSPHATASE (POTASSIUM SODIUM); AMYLOID BETA PROTEIN; BASIC FIBROBLAST GROWTH FACTOR; HYDROGEN PEROXIDE; OUABAIN; REACTIVE OXYGEN METABOLITE; SYNTHETIC PEPTIDE;

ANIMAL CELL; ARTICLE; BRAIN CELL; CONTROLLED STUDY; EMBRYO; ENZYME ACTIVITY; HIPPOCAMPUS; LIPID PEROXIDATION; MEMBRANE POTENTIAL; MITOCHONDRIAL MEMBRANE; NERVE CELL CULTURE; NERVE CELL DEGENERATION; NEUROPROTECTION; NEUROTOXICITY; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; RAT;

AMYLOID BETA-PROTEIN; ANIMALS; FIBROBLAST GROWTH FACTOR 2; HIPPOCAMPUS; HYDROGEN PEROXIDE; LIPID PEROXIDES; MITOCHONDRIA; NA(+)-K(+)-EXCHANGING ATPASE; NEURONS; NEUROTOXINS; OXIDATIVE STRESS; RATS;

EID: 0030902464     PISSN: 00068993     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0006-8993(97)00196-0     Document Type: Article

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