Behavioral defects in C. elegans egl-36 mutants result from potassium channels shifted in voltage-dependence of activation

Neuron

Volumn 19, Issue 1, 1997, Pages 151-164

  Johnstone, Duncan B ^a   Wei, Aguan ^b   Butler, Alice ^b   Salkoff, Lawrence ^b   Thomas, James H ^a  

^a UNIVERSITY OF WASHINGTON   (United States)

^b WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

POTASSIUM; POTASSIUM CHANNEL;

AMINO ACID SUBSTITUTION; AMINO TERMINAL SEQUENCE; ANIMAL BEHAVIOR; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CAENORHABDITIS ELEGANS; CHANNEL GATING; CONTROLLED STUDY; EGG LAYING; FEMALE; GENE MUTATION; INTESTINE MUSCLE; MALE; MUTANT; NONHUMAN; OOCYTE; POTASSIUM CONDUCTANCE; PRIORITY JOURNAL; XENOPUS LAEVIS;

EID: 0030762007     PISSN: 08966273     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0896-6273(00)80355-4     Document Type: Article

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