Suppression of transforming growth factor beta/Smad signaling in keloid-derived fibroblasts by quercetin: Implications for the treatment of excessive scars

Journal of Trauma - Injury, Infection and Critical Care

Volumn 57, Issue 5, 2004, Pages 1032-1037

  Phan, Toan Thang ^a,b,c,d   Lim, Ivor Jiun ^b   Chan, Sui Yung ^b   Tan, Ee Kim ^b   Lee, Seng Teik ^a   Longaker, Michael T ^b  

^a SINGAPORE GENERAL HOSPITAL   (Singapore)

^b NATIONAL UNIVERSITY OF SINGAPORE   (Singapore)

^c STANFORD UNIVERSITY   (United States)

^d STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Fibroblasts; Keloid; Quercetin; Smad; TGF

Indexed keywords

COLLAGEN; QUERCETIN; SMAD PROTEIN; SMAD2 PROTEIN; SMAD3 PROTEIN; SMAD4 PROTEIN; SOMATOMEDIN C; TRANSFORMING GROWTH FACTOR BETA;

ANTINEOPLASTIC ACTIVITY; ARTICLE; CELL PROLIFERATION; COMPLEX FORMATION; CONCENTRATION RESPONSE; CONTROLLED STUDY; DRUG EFFECT; EXTRACELLULAR MATRIX; FIBROBLAST; HUMAN; HUMAN CELL; IMMUNOBLOTTING; KELOID; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; SIGNAL TRANSDUCTION;

ANTIOXIDANTS; BLOTTING, WESTERN; CELL PROLIFERATION; CELLS, CULTURED; CICATRIX; DNA-BINDING PROTEINS; FIBROBLASTS; HUMANS; IMMUNOPRECIPITATION; KELOID; QUERCETIN; RECEPTORS, TRANSFORMING GROWTH FACTOR BETA; SMAD PROTEINS; TRANS-ACTIVATORS;

EID: 9644263886     PISSN: 00225282     EISSN: None     Source Type: Journal    
DOI: 10.1097/01.TA.0000114087.46566.EB     Document Type: Article

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