β-cell-targeted expression of a dominant-negative mutant of hepatocyte nuclear factor-1α in mice: Diabetes model with β-cell dysfunction partially rescued by nonglucose secretagogues

Diabetes

Volumn 53, Issue SUPPL. 3, 2004, Pages

  Winzell, Maria Sörhede ^a   Pacini, Giovanni ^b   Wollheim, Claes B ^c   Ahrén, Bo ^a  

^a LUND UNIVERSITY   (Sweden)

^b CNR   (Italy)

^c UNIVERSITY OF GENEVA MEDICAL SCHOOL   (Switzerland)

Author keywords

[No Author keywords available]

Indexed keywords

ARGININE; CARBACHOL; INSULIN; NUCLEAR FACTOR I; NUCLEAR FACTOR IALPHA; PALMITIC ACID; POTASSIUM CHLORIDE; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; CELL FUNCTION; CONFERENCE PAPER; CONTROLLED STUDY; DEPOLARIZATION; DIABETES MELLITUS; FEMALE; GENE EXPRESSION; GLUCOSE INTOLERANCE; INSULIN RESPONSE; INTRAVENOUS GLUCOSE TOLERANCE TEST; MODEL; MOUSE; MUTANT; NONHUMAN; PANCREAS ISLET BETA CELL; PRIORITY JOURNAL; TARGET CELL; TRANSGENIC MOUSE; WILD TYPE;

ANIMALS; ARGININE; BLOOD GLUCOSE; CARBACHOL; DNA-BINDING PROTEINS; GENES, DOMINANT; GLUCOSE; HEPATOCYTE NUCLEAR FACTOR 1; HEPATOCYTE NUCLEAR FACTOR 1-ALPHA; INSULIN; ISLETS OF LANGERHANS; MALE; MICE; MICE, TRANSGENIC; MUTATION; NUCLEAR PROTEINS; PALMITIC ACID; POTASSIUM CHLORIDE; RATS; TRANSCRIPTION FACTORS;

EID: 9444219603     PISSN: 00121797     EISSN: None     Source Type: Journal    
DOI: 10.2337/diabetes.53.suppl_3.S92     Document Type: Conference Paper

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