Characterization of CD38 in the major cell types of the heart: Endothelial cells highly express CD38 with activation by hypoxia-reoxygenation triggering NAD(P)H depletion

American Journal of Physiology - Cell Physiology

Volumn 314, Issue 3, 2018, Pages C297-C309

  Boslett, James ^a   Hemann, Craig ^a   Christofi, Fedias L ^b   Zweier, Jay L ^a  

^a OHIO STATE UNIVERSITY   (United States)

^b THE OHIO STATE UNIVERSITY WEXNER MEDICAL CENTER   (United States)

Author keywords

Endothelium; ENOS; Ischemia reperfusion; NAD(P)(H); Nitric oxide; Superoxide

Indexed keywords

ADP RIBOSYL CYCLASE/CYCLIC ADP RIBOSE HYDROLASE 1; ENDOTHELIAL NITRIC OXIDE SYNTHASE; MESSENGER RNA; NITRIC OXIDE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE; SUPEROXIDE; ADP RIBOSYL CYCLASE; CD38 PROTEIN, MOUSE; CD38 PROTEIN, RAT; MEMBRANE PROTEIN; NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE; NOS3 PROTEIN, MOUSE; NOS3 PROTEIN, RAT;

ANIMAL CELL; ARTICLE; CARDIAC MUSCLE CELL; CONTROLLED STUDY; ENDOTHELIAL DYSFUNCTION; ENDOTHELIUM CELL; ENZYME ACTIVITY; FIBROBLAST; GENE SILENCING; GENETIC TRANSCRIPTION; HYPOXIA; IMAGING; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; PROTEIN ANALYSIS; PROTEIN EXPRESSION; PROTEIN FUNCTION; RAT; REOXYGENATION; ANIMAL; C57BL MOUSE; CELL HYPOXIA; COMPARATIVE STUDY; CORONARY BLOOD VESSEL; DEFICIENCY; ENZYME ACTIVATION; ENZYMOLOGY; GENETICS; HEART MUSCLE REPERFUSION; KNOCKOUT MOUSE; METABOLISM; MYOCARDIAL ISCHEMIA REPERFUSION INJURY; PATHOLOGY; SIGNAL TRANSDUCTION; SPRAGUE DAWLEY RAT; TIME FACTOR;

ADP-RIBOSYL CYCLASE; ADP-RIBOSYL CYCLASE 1; ANIMALS; CELL HYPOXIA; CORONARY VESSELS; ENDOTHELIAL CELLS; ENZYME ACTIVATION; FIBROBLASTS; MEMBRANE GLYCOPROTEINS; MICE, INBRED C57BL; MICE, KNOCKOUT; MYOCARDIAL REPERFUSION; MYOCARDIAL REPERFUSION INJURY; MYOCYTES, CARDIAC; NADP; NITRIC OXIDE; NITRIC OXIDE SYNTHASE TYPE III; RATS, SPRAGUE-DAWLEY; SIGNAL TRANSDUCTION; SUPEROXIDES; TIME FACTORS;

EID: 85043587580     PISSN: 03636143     EISSN: 15221563     Source Type: Journal    
DOI: 10.1152/ajpcell.00139.2017     Document Type: Article

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