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Volumn 4, Issue 1, 2017, Pages

Mutant p53–Nrf2 axis regulates the proteasome machinery in cancer

Author keywords

APR 246; cancer; Chip seq; Keap1; multi omics; mutant p53; Nrf2; proteasome; proteomics; TNBC; transcriptomics

Indexed keywords


EID: 85041963321     PISSN: None     EISSN: 23723556     Source Type: Journal    
DOI: 10.1080/23723556.2016.1217967     Document Type: Article
Times cited : (15)

References (10)
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    • The dual roles of NRF2 in cancer
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    • Menegon S, Columbano A, Giordano S. The dual roles of NRF2 in cancer. Trends Mol Med 2016; xx:1-16; PMID:27263465; http://dx.doi.org/10.1016/j.molmed.2016.05.002
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    • Hayes JD, Dinkova-Kostova AT. The Nrf2 regulatory network provides an interface between redox and intermediary metabolism. Trends Biochem Sci 2014; 39:199-218; PMID:24647116; http://dx.doi.org/10.1016/j.tibs.2014.02.002
    • (2014) Trends Biochem Sci , vol.39 , pp. 199-218
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  • 8
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    • Modulation of oxidative stress as an anticancer strategy
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    • Gorrini, C.1    Harris, I.S.2    Mak, T.W.3
  • 9
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    • Mutant p53R273H attenuates the expression of phase 2 detoxifying enzymes and promotes the survival of cells with high levels of reactive oxygen species
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    • Kalo E, Kogan-Sakin I, Solomon H, Bar-Nathan E, Shay M, Shetzer Y, Dekel E, Goldfinger N, Buganim Y, Stambolsky P, et al. Mutant p53R273H attenuates the expression of phase 2 detoxifying enzymes and promotes the survival of cells with high levels of reactive oxygen species. J Cell Sci 2012; 125:5578-86; PMID:22899716; http://dx.doi.org/10.1242/jcs.106815
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    • Kalo, E.1    Kogan-Sakin, I.2    Solomon, H.3    Bar-Nathan, E.4    Shay, M.5    Shetzer, Y.6    Dekel, E.7    Goldfinger, N.8    Buganim, Y.9    Stambolsky, P.10
  • 10
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    • Targeting of mutant p53 and the cellular redox balance by APR-246 as a strategy for efficient cancer therapy
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    • Bykov VJN, Zhang Q, Zhang M, Ceder S, Abrahmsen L, Wiman KG. Targeting of mutant p53 and the cellular redox balance by APR-246 as a strategy for efficient cancer therapy. Front Oncol 2016; 6:21; PMID:26870698; http://dx.doi.org/10.3389/fonc.2016.00021
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.