JS-K promotes apoptosis by inducing ROS production in human prostate cancer cells

Oncology Letters

Volumn 13, Issue 3, 2017, Pages 1137-1142

  Qiu, Mingning ^a   Chen, Lieqian ^a   Tan, Guobin ^a   Ke, Longzhi ^a   Zhang, Sai ^a   Chen, Hege ^a   Liu, Jianjun ^a  

^a GUANGDONG MEDICAL UNIVERSITY   (China)

Author keywords

Apoptosis; JS K; Nitric oxide; Prostate cancer cells; Reactive oxygen species

Indexed keywords

ADENOSINE TRIPHOSPHATE; ANTINEOPLASTIC AGENT; CASPASE 9; GLUTATHIONE DISULFIDE; GLUTATHIONE TRANSFERASE; JS K DRUG; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE; NITRIC OXIDE; NITRIC OXIDE DONOR; PRODRUG; PROTEIN BAK; PROTEIN BAX; PROTEIN BCL 2; REACTIVE NITROGEN SPECIES; REACTIVE OXYGEN METABOLITE; SUPEROXIDE; UNCLASSIFIED DRUG;

APOPTOSIS; APOPTOSIS ASSAY; ARTICLE; CANCER GROWTH; CANCER INHIBITION; CELL PROLIFERATION ASSAY; CONTROLLED STUDY; DOSE RESPONSE; DOWN REGULATION; FLOW CYTOMETRY; GENE EXPRESSION; HUMAN; HUMAN CELL; MITOCHONDRIAL MEMBRANE POTENTIAL; PROSTATE CANCER; PROSTATE CANCER CELL LINE; RADIOIMMUNOPRECIPITATION; TIME-DEPENDENT INHIBITION; UPREGULATION; WESTERN BLOTTING;

EID: 85011880120     PISSN: 17921074     EISSN: 17921082     Source Type: Journal    
DOI: 10.3892/ol.2016.5535     Document Type: Article

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