Core 1- and 3-derived O-glycans collectively maintain the colonic mucus barrier and protect against spontaneous colitis in mice

Mucosal Immunology

Volumn 10, Issue 1, 2017, Pages 91-103

  Bergstrom, K ^a   Fu, J ^a,b   Johansson, M E V ^c   Liu, X ^d   Gao, N ^b   Wu, Q ^a,e   Song, J ^a   McDaniel, J M ^a   McGee, S ^a   Chen, W ^b   Braun, J ^f   Hansson, G C ^c   Xia, L ^a,b,g  

^a OKLAHOMA MEDICAL RESEARCH FOUNDATION   (United States)

^b THE FIRST AFFILIATED HOSPITAL OF SOOCHOW UNIVERSITY   (China)

^c UNIVERSITY OF GOTHENBURG   (Sweden)

^d CENTRAL SOUTH UNIVERSITY   (China)

^e FUDAN UNIVERSITY   (China)

^f UNIVERSITY OF CALIFORNIA   (United States)

^g UNIVERSITY OF OKLAHOMA COLLEGE OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AMPICILLIN; CORE 1 BETA1,3 GALACTOSYLTRANSFERASE; CORE 1 DERIVED O GLYCAN; CORE 3 BETA1,3 N ACETYLGLUCOSAMINYLTRANSFERASE; CORE 3 DERIVED O GLYCAN; GALACTOSYLTRANSFERASE; METRONIDAZOLE; MUCIN 2; NEOMYCIN; OLIGOSACCHARIDE; PROTEINASE; TRANSFERASE; UNCLASSIFIED DRUG; VANCOMYCIN; ANTIINFECTIVE AGENT; BETA-1,3-GALACTOSYL-O-GLYCOSYL-GLYCOPROTEIN BETA-1,6-N-ACETYLGLUCOSAMINYLTRANSFERASE 3; C1GALT1 PROTEIN, MOUSE; N ACETYLGLUCOSAMINYLTRANSFERASE; POLYSACCHARIDE;

ADULT; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; ASCENDING COLON; COLITIS; CONTROLLED STUDY; DESCENDING COLON; DISEASE PREDISPOSITION; DISEASE SEVERITY; ENZYME DEGRADATION; FEMALE; GLYCOSYLATION; INTESTINE FLORA; MALE; MOUSE; MUCUS; NONHUMAN; PROTEIN DEGRADATION; PROTEIN EXPRESSION; WILD TYPE; ANIMAL; C57BL MOUSE; CELL CULTURE; COLITIS, ULCERATIVE; COLON; DISEASE MODEL; DRUG EFFECTS; GENETICS; HUMAN; IMMUNOLOGY; INTESTINE MUCOSA; KNOCKOUT MOUSE; METABOLISM; MUCOSAL IMMUNITY; PATHOLOGY;

ANIMALS; ANTI-BACTERIAL AGENTS; CELLS, CULTURED; COLITIS, ULCERATIVE; COLON; DISEASE MODELS, ANIMAL; GALACTOSYLTRANSFERASES; HUMANS; IMMUNITY, MUCOSAL; INTESTINAL MUCOSA; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MUCUS; N-ACETYLGLUCOSAMINYLTRANSFERASES; POLYSACCHARIDES; PROTEOLYSIS;

EID: 85011085111     PISSN: 19330219     EISSN: 19353456     Source Type: Journal    
DOI: 10.1038/mi.2016.45     Document Type: Article

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