Cellular defects by deletion of ANO10 are due to deregulated local calcium signaling

Cellular Signalling

Volumn 30, Issue , 2017, Pages 41-49

  Wanitchakool, Podchanart ^a   Ousingsawat, Jiraporn ^a   Sirianant, Lalida ^a   Cabrita, Inês ^a   Faria, Diana ^a   Schreiber, Rainer ^a   Kunzelmann, Karl ^a  

^a UNIVERSITY OF REGENSBURG   (Germany)

Author keywords

ANO10; Anoctamin 10; Apoptosis; Ca2 + signaling; Ion channels; Jejunum; Macrophages; TMEM16K

Indexed keywords

ANO10 PROTEIN; AXS PROTEIN; CALCIUM CHANNEL; TUBULIN; TUMOR NECROSIS FACTOR; UNCLASSIFIED DRUG; ANOCTAMIN; CASPASE 3;

ANIMAL CELL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CALCIUM SIGNALING; CELL CYCLE; CELL DAMAGE; CELL SIZE; CENTRIOLE; CONTROLLED STUDY; ENDOPLASMIC RETICULUM; GENE INACTIVATION; HUMAN; HUMAN CELL; INTESTINE EPITHELIUM CELL; ION TRANSPORT; MITOSIS SPINDLE; MOUSE; NONHUMAN; PERITONEUM MACROPHAGE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN LOCALIZATION; RAT; THYROID CELL; ANIMAL; CELL PROLIFERATION; CYTOLOGY; DEFICIENCY; GENE DELETION; HEK293 CELL LINE; INTESTINE CELL; KNOCKOUT MOUSE; MACROPHAGE; METABOLISM; PROTEIN TRANSPORT;

ANIMALS; ANOCTAMINS; APOPTOSIS; CALCIUM SIGNALING; CASPASE 3; CELL PROLIFERATION; CELL SIZE; ENTEROCYTES; GENE DELETION; HEK293 CELLS; HUMANS; MACROPHAGES; MICE, KNOCKOUT; PROTEIN TRANSPORT; RATS;

EID: 84996721484     PISSN: 08986568     EISSN: 18733913     Source Type: Journal    
DOI: 10.1016/j.cellsig.2016.11.006     Document Type: Article

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