Elucidating the effects of disease-causing mutations on STAT3 function in autosomal-dominant hyper-IgE syndrome

Journal of Allergy and Clinical Immunology

Volumn 138, Issue 4, 2016, Pages 1210-1213.e5

  Pelham, Simon J ^a,b   Lenthall, Helen C ^b   Deenick, Elissa K ^a,b   Tangye, Stuart G ^a,b  

^a GARVAN INSTITUTE OF MEDICAL RESEARCH   (Australia)

^b UNIVERSITY OF NEW SOUTH WALES   (Australia)

Author keywords

[No Author keywords available]

Indexed keywords

DNA; IMMUNOGLOBULIN E; INTERLEUKIN 10; INTERLEUKIN 21; INTERLEUKIN 23; INTERLEUKIN 6; PROTEIN SH2; STAT3 PROTEIN; TRANSCRIPTION FACTOR; MUTANT PROTEIN; RECOMBINANT PROTEIN; STAT3 PROTEIN, HUMAN;

ANTIBODY PRODUCTION; ARTICLE; AUTOSOMAL DOMINANT INHERITANCE; CONTROLLED STUDY; DNA BINDING; GENE MUTATION; GENETIC TRANSCRIPTION; HEK293 CELL LINE; HUMAN; HUMAN CELL; HYPER IGE SYNDROME; MEMORY CELL; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; STEREOSPECIFICITY; TH17 CELL; TRANSACTIVATION; AMINO ACID SUBSTITUTION; CHEMISTRY; DOMINANT GENE; GENETICS; IMMUNOLOGY; METABOLISM; MUTATION; PHOSPHORYLATION; PROTEIN MULTIMERIZATION;

AMINO ACID SUBSTITUTION; GENES, DOMINANT; HEK293 CELLS; HUMANS; JOB SYNDROME; MUTANT PROTEINS; MUTATION; PHOSPHORYLATION; PROTEIN MULTIMERIZATION; RECOMBINANT PROTEINS; STAT3 TRANSCRIPTION FACTOR;

EID: 84992208947     PISSN: 00916749     EISSN: 10976825     Source Type: Journal    
DOI: 10.1016/j.jaci.2016.04.020     Document Type: Article

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