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Volumn 21, Issue 3, 2016, Pages 305-317

Exposure of aconitase to smoking-related oxidants results in iron loss and increased iron response protein-1 activity: potential mechanisms for iron accumulation in human arterial cells

Author keywords

Aconitase; Cardiovascular disease; Hypothiocyanous acid; Inflammation; Iron release; Iron response protein 1; Myeloperoxidase; Thiol oxidation

Indexed keywords

ACONITATE HYDRATASE; CYSTEINE; FERRITIN; FERROUS SULFIDE; HYPOTHIOCYANOUS ACID; IRON; IRON RESPONSE PROTEIN 1; ISOPROTEIN; MYELOPEROXIDASE; PROTEIN; THIOCYANATE; THIOL; UNCLASSIFIED DRUG; HYPOCHLOROUS ACID; IRON REGULATORY FACTOR; OXIDIZING AGENT; THIOCYANIC ACID DERIVATIVE;

EID: 84989332527     PISSN: 09498257     EISSN: 14321327     Source Type: Journal    
DOI: 10.1007/s00775-016-1340-4     Document Type: Article
Times cited : (9)

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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.