Blockade of receptor for advanced glycation end products protects against systolic overload-induced heart failure after transverse aortic constriction in mice

European Journal of Pharmacology

Volumn 791, Issue , 2016, Pages 535-543

  Liu, Yu ^a,b   Yu, Manli ^b   Zhang, Zhigang ^b   Yu, Yunhua ^b   Chen, Qi ^b   Zhang, Wei ^b   Zhao, Xianxian ^b  

^a NANJING UNIVERSITY   (China)

^b SECOND MILITARY MEDICAL UNIVERSITY   (China)

Author keywords

AMPK mTOR; Heart failure; NF B; Receptor for advanced glycation end products; Transverse aortic constriction

Indexed keywords

ADVANCED GLYCATION END PRODUCT RECEPTOR; ADVANCED GLYCATION END PRODUCT RECEPTOR ANTAGONIST; FPS ZM 1; MAMMALIAN TARGET OF RAPAMYCIN; MESSENGER RNA; TRANSCRIPTION FACTOR RELA; UNCLASSIFIED DRUG; ADVANCED GLYCATION END PRODUCT; AGER PROTEIN, MOUSE; BENZAMIDE DERIVATIVE; FPS-ZM1; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; TARGET OF RAPAMYCIN KINASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; AORTA CONSTRICTION; ARTICLE; DRUG RECEPTOR BINDING; ENDOPLASMIC RETICULUM STRESS; ENZYME INHIBITION; HEART MUSCLE FIBROSIS; HEART VENTRICLE HYPERTROPHY; HEART VENTRICLE OVERLOAD; LIMIT OF QUANTITATION; MALE; MOUSE; NONHUMAN; OXIDATIVE STRESS; PATHOGENESIS; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; TRANSVERSE AORTIC CONSTRICTION; UPREGULATION; ADVERSE EFFECTS; ANIMAL; ANTAGONISTS AND INHIBITORS; AORTA; C57BL MOUSE; CHEMISTRY; COMPLICATION; DRUG EFFECTS; FIBROSIS; HEART FAILURE; HYPERTROPHY; METABOLISM; PATHOLOGY; PATHOPHYSIOLOGY; PRESSURE; SIGNAL TRANSDUCTION; SOLUBILITY; SYSTOLE; VASOCONSTRICTION;

ADVANCED GLYCOSYLATION END PRODUCT-SPECIFIC RECEPTOR; ANIMALS; AORTA; BENZAMIDES; ENDOPLASMIC RETICULUM STRESS; FIBROSIS; GLYCOSYLATION END PRODUCTS, ADVANCED; HEART FAILURE; HYPERTROPHY; MALE; MICE; MICE, INBRED C57BL; NF-KAPPA B; OXIDATIVE STRESS; PRESSURE; SIGNAL TRANSDUCTION; SOLUBILITY; SYSTOLE; TOR SERINE-THREONINE KINASES; VASOCONSTRICTION;

EID: 84988646859     PISSN: 00142999     EISSN: 18790712     Source Type: Journal    
DOI: 10.1016/j.ejphar.2016.07.008     Document Type: Article

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