MCT8 deficiency in male mice mitigates the phenotypic abnormalities associated with the absence of a functional type 3 deiodinase

Endocrinology

Volumn 157, Issue 8, 2016, Pages 3266-3277

  Stohn, J Patrizia ^a   Martinez, M Elena ^a   Matoin, Kassey ^a   Morte, Beatriz ^b   Bernal, Juan ^b   Galton, Valerie Anne ^c   Germain, Donald St ^a   Hernandez, Arturo ^a  

^a Maine Medical Center Research Institute   (United States)

^b CSIC   (Spain)

^c DARTMOUTH MEDICAL SCHOOL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

DEIODINASE TYPE 3; LIOTHYRONINE; MONOCARBOXYLATE TRANSPORTER 8; OXIDOREDUCTASE; THYROTROPIN; THYROXINE; UNCLASSIFIED DRUG; CARRIER PROTEIN; IODIDE PEROXIDASE; IODOTHYRONINE DEIODINASE TYPE III; SLC16A2 PROTEIN, MOUSE;

ADULT; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; GENE EXPRESSION; GENOTYPE; GROWTH RETARDATION; HISTOLOGY; HYPOTHALAMUS; HYPOTHYROIDISM; LETHALITY; LIOTHYRONINE BLOOD LEVEL; MALE; MONOCARBOXYLATE TRANSPORTER 8 DEFICIENCY; MOUSE; NEOCORTEX; NEWBORN; NONHUMAN; ONTOGENY; PERINATAL MORTALITY; PHENOTYPE; PRIORITY JOURNAL; PROTEIN DEFICIENCY; PROTEIN EXPRESSION; REAL TIME POLYMERASE CHAIN REACTION; TANDEM MASS SPECTROMETRY; THYROID GLAND; THYROID WEIGHT; THYROTOXICOSIS; THYROTROPIN BLOOD LEVEL; ANIMAL; FETAL WELL BEING; GENETICS; GROWTH, DEVELOPMENT AND AGING; INTRAUTERINE GROWTH RETARDATION; KNOCKOUT MOUSE; PHYSIOLOGY;

ANIMALS; ANIMALS, NEWBORN; FETAL GROWTH RETARDATION; FETAL VIABILITY; GROWTH AND DEVELOPMENT; HYPOTHALAMUS; HYPOTHYROIDISM; IODIDE PEROXIDASE; MALE; MEMBRANE TRANSPORT PROTEINS; MICE; MICE, KNOCKOUT; PHENOTYPE; THYROID GLAND;

EID: 84982803309     PISSN: 00137227     EISSN: 19457170     Source Type: Journal    
DOI: 10.1210/en.2016-1162     Document Type: Article

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