TRAF3IP2 mediates aldosterone/salt-induced cardiac hypertrophy and fibrosis

Molecular and Cellular Endocrinology

Volumn 429, Issue , 2016, Pages 84-92

  Sakamuri, Siva S V P ^a,e   Valente, Anthony J ^b   Siddesha, Jalahalli M ^a,f   Delafontaine, Patrice ^a,g   Siebenlist, Ulrich ^c   Gardner, Jason D ^d   Bysani, Chandrasekar ^a,h  

^a TULANE UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b UNIVERSITY OF TEXAS HEALTH SCIENCE CENTER AT SAN ANTONIO   (United States)

^c NATIONAL INSTITUTE OF ALLERGY AND INFECTIOUS DISEASES   (United States)

^d LOUISIANA STATE UNIVERSITY HEALTH SCIENCES CENTER   (United States)

^e UNIVERSITY OF ALBERTA   (Canada)

^f UNIVERSITY OF VERMONT COLLEGE OF MEDICINE   (United States)

^g University of Missouri   (United States)

^h VETERANS AFFAIRS MEDICAL CENTER   (United States)

Author keywords

Act1; Aldosterone; Cardiac fibrosis; Cardiac hypertrophy; CIKS; RAAS; TRAF3IP2

Indexed keywords

ALDOSTERONE; COLLAGEN; COLLAGEN 1A1; COLLAGEN 3A1; CXCL1 CHEMOKINE; CXCL2 CHEMOKINE; DRINKING WATER; GELATINASE A; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; INTERCELLULAR ADHESION MOLECULE 1; INTERLEUKIN 18; INTERLEUKIN 6; MESSENGER RNA; PROTEIN C JUN; PROTEIN LYSINE 6 OXIDASE; SODIUM CHLORIDE; SYNAPTOTAGMIN I; TRANSCRIPTION FACTOR AP 1; UNCLASSIFIED DRUG; ANGIOTENSIN 1 RECEPTOR; AUTACOID; CELL ADHESION MOLECULE; CYTOKINE; SIGNAL TRANSDUCING ADAPTOR PROTEIN; TRAF3IP2 PROTEIN, MOUSE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; ELEVATED BLOOD PRESSURE; ENZYME ACTIVATION; GENE; GENE DELETION; GENE EXPRESSION; HEART MUSCLE FIBROSIS; HEART VENTRICLE HYPERTROPHY; IN VIVO STUDY; MALE; MOUSE; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; TRAF3IP2 GENE; UPREGULATION; ANIMAL; BLOOD PRESSURE; C57BL MOUSE; CARDIOMEGALY; CHEMICALLY INDUCED; DIAGNOSTIC IMAGING; DRUG EFFECTS; EXTRACELLULAR MATRIX; FIBROSIS; GENE EXPRESSION REGULATION; GENETICS; METABOLISM;

ADAPTOR PROTEINS, SIGNAL TRANSDUCING; ALDOSTERONE; ANIMALS; BLOOD PRESSURE; CARDIOMEGALY; CELL ADHESION MOLECULES; CYTOKINES; EXTRACELLULAR MATRIX; FIBROSIS; GENE EXPRESSION REGULATION; INFLAMMATION MEDIATORS; MATRIX METALLOPROTEINASE 2; MICE, INBRED C57BL; NF-KAPPA B; RECEPTOR, ANGIOTENSIN, TYPE 1; SODIUM CHLORIDE; TRANSCRIPTION FACTOR AP-1;

EID: 84975524950     PISSN: 03037207     EISSN: 18728057     Source Type: Journal    
DOI: 10.1016/j.mce.2016.03.038     Document Type: Article

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