Pendrin and anoctamin as mediators of apical iodide efflux in thyroid cells

Current Opinion in Endocrinology, Diabetes and Obesity

Volumn 22, Issue 5, 2015, Pages 374-380

  Silveira, Jamile C ^a,b   Kopp, Peter A ^a  

^a NORTHWESTERN UNIVERSITY   (United States)

^b UNIVERSITY OF SÃO PAULO   (Brazil)

Author keywords

ANO 1 TMEM16A; Anoctamin 1; PDS SLC26A4; pendred syndrome; pendrin; thyroid hormone synthesis

Indexed keywords

ANOCTAMIN; PDS PROTEIN; PENDRIN; PROTEIN; UNCLASSIFIED DRUG; ANO1 PROTEIN, HUMAN; CARRIER PROTEIN; CHLORIDE CHANNEL; IODIDE; SLC26A4 PROTEIN, HUMAN; THYROID HORMONE; TUMOR PROTEIN;

APICAL IODIDE EFFLUX; APICAL MEMBRANE; EFFUSION; GENE MUTATION; HUMAN; NONHUMAN; PENDRED SYNDROME; PHENOTYPE; REVIEW; THYROID CELL; ANIMAL; BIOSYNTHESIS; CYTOLOGY; GENETICS; METABOLISM; NODULAR GOITER; PERCEPTION DEAFNESS; PHYSIOLOGY; THYROID GLAND;

ANIMALS; CHLORIDE CHANNELS; GOITER, NODULAR; HEARING LOSS, SENSORINEURAL; HUMANS; IODIDES; MEMBRANE TRANSPORT PROTEINS; NEOPLASM PROTEINS; THYROID GLAND; THYROID HORMONES;

EID: 84960116515     PISSN: 1752296X     EISSN: 17522978     Source Type: Journal    
DOI: 10.1097/MED.0000000000000188     Document Type: Review

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