Role of cytosolic and calcium independent phospholipases A2 in insulin secretion impairment of INS-1E cells infected by S. aureus

FEBS Letters

Volumn 589, Issue 24, 2015, Pages 3969-3976

  Caporarello, N ^a   Salmeri, M ^a   Scalia, M ^a   Motta, C ^a   Parrino, C ^a   Frittitta, L ^a   Olivieri, M ^a   Toscano, M A ^a   Anfuso, C D ^a   Lupo, G ^a  

^a UNIVERSITY OF CATANIA   (Italy)

Author keywords

INS 1E cell; Insulin; Phospholipases A2; siRNA; Staphylococcus aureus

Indexed keywords

CALCIUM INDEPENDENT PHOSPHOLIPASE A2; CYCLOOXYGENASE 2; CYCLOOXYGENASE 2 INHIBITOR; CYTOSOLIC PHOSPHOLIPASE A2; MITOGEN ACTIVATED PROTEIN KINASE 1; MITOGEN ACTIVATED PROTEIN KINASE 3; SMALL INTERFERING RNA; INSULIN; PHOSPHOLIPASE A2 GROUP IV; PHOSPHOLIPASE A2 GROUP VI; PLA2G4A PROTEIN, RAT; PLA2G6 PROTEIN, RAT; PROTEIN KINASE C ALPHA; PTGS2 PROTEIN, RAT;

ANIMAL CELL; ARTICLE; CELL LINE; CONTROLLED STUDY; ENZYME ACTIVITY; ENZYME PHOSPHORYLATION; INS 1E CELL LINE; INSULIN RELEASE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; STAPHYLOCOCCUS INFECTION; ANIMAL; ANTAGONISTS AND INHIBITORS; CHEMISTRY; DIABETES MELLITUS, TYPE 1; DRUG EFFECTS; ENZYMOLOGY; GENETICS; HOST PATHOGEN INTERACTION; KINETICS; METABOLISM; METHICILLIN RESISTANT STAPHYLOCOCCUS AUREUS; MICROBIOLOGY; PANCREAS ISLET BETA CELL; PANCREATITIS; PATHOPHYSIOLOGY; PHOSPHORYLATION; PHYSIOLOGY; PROTEIN PROCESSING; RNA INTERFERENCE; SECRETION (PROCESS); SIGNAL TRANSDUCTION; TUMOR CELL LINE;

ANIMALS; CELL LINE, TUMOR; CYCLOOXYGENASE 2; CYCLOOXYGENASE 2 INHIBITORS; DIABETES MELLITUS, TYPE 1; GROUP IV PHOSPHOLIPASES A2; GROUP VI PHOSPHOLIPASES A2; HOST-PATHOGEN INTERACTIONS; INSULIN; INSULIN-SECRETING CELLS; KINETICS; MAP KINASE SIGNALING SYSTEM; METHICILLIN-RESISTANT STAPHYLOCOCCUS AUREUS; PANCREATITIS; PHOSPHORYLATION; PROTEIN KINASE C-ALPHA; PROTEIN PROCESSING, POST-TRANSLATIONAL; RATS; RNA INTERFERENCE; STAPHYLOCOCCAL INFECTIONS;

EID: 84959548045     PISSN: 00145793     EISSN: 18733468     Source Type: Journal    
DOI: 10.1016/j.febslet.2015.11.035     Document Type: Article

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