RIP3-mediated necrotic cell death accelerates systematic inflammation and mortality

Proceedings of the National Academy of Sciences of the United States of America

Volumn 112, Issue 35, 2015, Pages 11007-11012

  Meng, Lingjun ^a,b   Jin, Wei ^c   Wang, Xiaodong ^a  

^a NATIONAL INSTITUTE OF BIOLOGICAL SCIENCES   (China)

^b CHINA AGRICULTURAL UNIVERSITY   (China)

^c TSINGHUA UNIVERSITY   (China)

Author keywords

Atherosclerosis; Longevity; Macrophages; Necrosis; RIP3

Indexed keywords

APOLIPOPROTEIN E; CYTOKINE; INTERLEUKIN 1ALPHA; LYMPHOCYTE ANTIGEN; LYMPHOCYTE ANTIGEN 6C; MESSENGER RNA; PROTEIN KINASE; RECEPTOR INTERACTING PROTEIN 3; UNCLASSIFIED DRUG; CHOLESTEROL INTAKE; RECEPTOR INTERACTING PROTEIN SERINE THREONINE KINASE; RIPK3 PROTEIN, MOUSE;

ADIPOCYTE; ADIPOSE TISSUE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; ATHEROSCLEROTIC PLAQUE; BLOOD SAMPLING; BONE MARROW; CONTROLLED STUDY; CYTOKINE RELEASE; INFLAMMATION; LIFESPAN; LYMPHOCYTIC INFILTRATION; MALE; MEDIATOR; MONOCYTE; MORTALITY; MOUSE; NECROPTOSIS; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; SKIN DEFECT; ADMINISTRATION AND DOSAGE; ANIMAL; C57BL MOUSE; CELL DEATH; CHOLESTEROL INTAKE; GENETICS; KNOCKOUT MOUSE; PATHOPHYSIOLOGY; PHYSIOLOGY;

ANIMALS; APOLIPOPROTEINS E; CELL DEATH; CHOLESTEROL, DIETARY; INFLAMMATION; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; RECEPTOR-INTERACTING PROTEIN SERINE-THREONINE KINASES;

EID: 84940992105     PISSN: 00278424     EISSN: 10916490     Source Type: Journal    
DOI: 10.1073/pnas.1514730112     Document Type: Article

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