ERα inhibits epithelial-mesenchymal transition by suppressing Bmi1 in breast cancer

Oncotarget

Volumn 6, Issue 25, 2015, Pages 21704-21717

  Wei, Xiao Long ^a,b   Dou, Xiao Wei ^b   Bai, Jing Wen ^b   Luo, Xiang Rong ^b   Qiu, Si Qi ^a   Xi, Di Di ^b   Huang, Wen He ^a   Du, Cai Wen ^a   Man, Kwan ^c   Zhang, Guo Jun ^a,b  

^a CANCER HOSPITAL OF SHANTOU UNIVERSITY MEDICAL COLLEGE   (China)

^b SHANTOU UNIVERSITY MEDICAL COLLEGE   (China)

^c UNIVERSITY OF HONG KONG   (Hong Kong)

Author keywords

Bmi1; Breast cancer; Epithelial mesenchymal transition; ER signaling; Stemness

Indexed keywords

BMI1 PROTEIN; CD24 ANTIGEN; ESTROGEN RECEPTOR ALPHA; HERMES ANTIGEN; UVOMORULIN; BMI1 PROTEIN, HUMAN; CADHERIN; CD44 PROTEIN, HUMAN; ESTROGEN; ESTROGEN RECEPTOR ALPHA, HUMAN; PROTEIN BINDING;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BREAST CANCER; CANCER CELL; CELL INVASION; CELL MIGRATION; CELL POPULATION; CONTROLLED STUDY; DOWN REGULATION; EPITHELIAL MESENCHYMAL TRANSITION; FEMALE; HISTOPATHOLOGY; HUMAN; HUMAN CELL; HUMAN TISSUE; IMMUNOHISTOCHEMISTRY; IN VITRO STUDY; IN VIVO STUDY; MOUSE; NONHUMAN; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN PROTEIN INTERACTION; RECEPTOR UPREGULATION; TRANSCRIPTION REGULATION; ANIMAL; ANTAGONISTS AND INHIBITORS; BAGG ALBINO MOUSE; BREAST TUMOR; CELL MOTION; CYTOLOGY; GENE EXPRESSION REGULATION; METABOLISM; METASTASIS; NUDE MOUSE; PROMOTER REGION; SIGNAL TRANSDUCTION; STEM CELL; TUMOR INVASION; WOUND HEALING;

ANIMALS; ANTIGENS, CD24; ANTIGENS, CD44; BREAST NEOPLASMS; CADHERINS; CELL MOVEMENT; EPITHELIAL-MESENCHYMAL TRANSITION; ESTROGEN RECEPTOR ALPHA; ESTROGENS; FEMALE; GENE EXPRESSION REGULATION, NEOPLASTIC; HUMANS; IMMUNOHISTOCHEMISTRY; MICE; MICE, INBRED BALB C; MICE, NUDE; NEOPLASM INVASIVENESS; NEOPLASM METASTASIS; POLYCOMB REPRESSIVE COMPLEX 1; PROMOTER REGIONS, GENETIC; PROTEIN BINDING; SIGNAL TRANSDUCTION; STEM CELLS; WOUND HEALING;

EID: 84940734548     PISSN: None     EISSN: 19492553     Source Type: Journal    
DOI: 10.18632/oncotarget.3966     Document Type: Article

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