-
1
-
-
84892805731
-
Cancer statistics, 2014
-
Siegel R, Ma J, Zou Z, Jemal A. Cancer statistics, 2014. CA Cancer J Clin 2014; 64:9-29.
-
(2014)
CA Cancer J Clin
, vol.64
, pp. 9-29
-
-
Siegel, R.1
Ma, J.2
Zou, Z.3
Jemal, A.4
-
3
-
-
84864185989
-
Inherited variation at chromosome 12p13.33, including RAD52, influences the risk of squamous cell lung carcinoma
-
Shi J, Chatterjee N, Rotunno M, et al. Inherited variation at chromosome 12p13.33, including RAD52, influences the risk of squamous cell lung carcinoma. Cancer Discov 2012; 2:131-139.
-
(2012)
Cancer Discov
, vol.2
, pp. 131-139
-
-
Shi, J.1
Chatterjee, N.2
Rotunno, M.3
-
5
-
-
0035902544
-
Rad52 forms DNA repair and recombination centers during S phase
-
Lisby M, Rothstein R, Mortensen UH. Rad52 forms DNA repair and recombination centers during S phase. Proc Natl Acad Sci USA 2001; 98:8276-8282.
-
(2001)
Proc Natl Acad Sci USA
, vol.98
, pp. 8276-8282
-
-
Lisby, M.1
Rothstein, R.2
Mortensen, U.H.3
-
6
-
-
84903585835
-
Rare variants of large effect in BRCA2 and CHEK2 affect risk of lung cancer
-
Wang Y, McKay JD, Rafnar T, et al. Rare variants of large effect in BRCA2 and CHEK2 affect risk of lung cancer. Nat Genet 2014; 46:736-741.
-
(2014)
Nat Genet
, vol.46
, pp. 736-741
-
-
Wang, Y.1
McKay, J.D.2
Rafnar, T.3
-
7
-
-
84870379165
-
Molecular pathways: Understanding the role of Rad52 in homologous recombination for therapeutic advancement
-
Lok BH, Powell SN. Molecular pathways: Understanding the role of Rad52 in homologous recombination for therapeutic advancement. Clin Cancer Res 2012; 18:6400-6406.
-
(2012)
Clin Cancer Res
, vol.18
, pp. 6400-6406
-
-
Lok, B.H.1
Powell, S.N.2
-
8
-
-
79551658805
-
Rad52 inactivation is synthetically lethal with BRCA2 deficiency
-
Feng Z, Scott SP, Bussen W, et al. Rad52 inactivation is synthetically lethal with BRCA2 deficiency. Proc Natl Acad Sci USA 2011; 108:686-691.
-
(2011)
Proc Natl Acad Sci USA
, vol.108
, pp. 686-691
-
-
Feng, Z.1
Scott, S.P.2
Bussen, W.3
-
9
-
-
0031741863
-
Targeted inactivation of mouse RAD52 reduces homologous recombination but not resistance to ionizing radiation
-
Rijkers T, Van Den Ouweland J, Morolli B, et al. Targeted inactivation of mouse RAD52 reduces homologous recombination but not resistance to ionizing radiation. Mol Cell Biol 1998; 18:6423-6429.
-
(1998)
Mol Cell Biol
, vol.18
, pp. 6423-6429
-
-
Rijkers, T.1
Van Den Ouweland, J.2
Morolli, B.3
-
10
-
-
84880917274
-
RAD52 inactivation is synthetically lethal with deficiencies in BRCA1 and PALB2 in addition to BRCA2 through RAD51-mediated homologous recombination
-
Lok BH, Carley AC, Tchang B, Powell SN. RAD52 inactivation is synthetically lethal with deficiencies in BRCA1 and PALB2 in addition to BRCA2 through RAD51-mediated homologous recombination. Oncogene 2013; 32:3552-3558.
-
(2013)
Oncogene
, vol.32
, pp. 3552-3558
-
-
Lok, B.H.1
Carley, A.C.2
Tchang, B.3
Powell, S.N.4
-
11
-
-
77950341944
-
Genetic variants and risk of lung cancer in never smokers: A genome-wide association study
-
Li Y, Sheu CC, Ye Y, et al. Genetic variants and risk of lung cancer in never smokers: A genome-wide association study. Lancet Oncol 2010; 11:321-330.
-
(2010)
Lancet Oncol
, vol.11
, pp. 321-330
-
-
Li, Y.1
Sheu, C.C.2
Ye, Y.3
-
12
-
-
84866434919
-
Genomic landscape of non-small cell lung cancer in smokers and never-smokers
-
Govindan R, Ding L, Griffith M, et al. Genomic landscape of non-small cell lung cancer in smokers and never-smokers. Cell 2012; 150:1121-1134.
-
(2012)
Cell
, vol.150
, pp. 1121-1134
-
-
Govindan, R.1
Ding, L.2
Griffith, M.3
-
13
-
-
0035083738
-
Inactivation of both Rb and p53 pathways in mouse lung epithelial cell lines
-
McDoniels-Silvers AL, Herzog CR, Tyson FL, Malkinson AM, You M. Inactivation of both Rb and p53 pathways in mouse lung epithelial cell lines. Exp Lung Res 2001; 27:297-318.
-
(2001)
Exp Lung Res
, vol.27
, pp. 297-318
-
-
McDoniels-Silvers, A.L.1
Herzog, C.R.2
Tyson, F.L.3
Malkinson, A.M.4
You, M.5
-
14
-
-
0035504929
-
Overexpression of human RAD51 and RAD52 reduces double-strand break-induced homologous recombination in mammalian cells
-
Kim PM, Allen C, Wagener BM, Shen Z, Nickoloff JA. Overexpression of human RAD51 and RAD52 reduces double-strand break-induced homologous recombination in mammalian cells. Nucleic Acids Res 2001; 29:4352-4360.
-
(2001)
Nucleic Acids Res
, vol.29
, pp. 4352-4360
-
-
Kim, P.M.1
Allen, C.2
Wagener, B.M.3
Shen, Z.4
Nickoloff, J.A.5
-
15
-
-
56149126490
-
C-MYC overexpression is required for continuous suppression of oncogene-induced senescence in melanoma cells
-
Zhuang D, Mannava S, Grachtchouk V, et al. C-MYC overexpression is required for continuous suppression of oncogene-induced senescence in melanoma cells. Oncogene 2008; 27:6623-6634.
-
(2008)
Oncogene
, vol.27
, pp. 6623-6634
-
-
Zhuang, D.1
Mannava, S.2
Grachtchouk, V.3
-
16
-
-
66349084196
-
H2AX is required for cell cycle arrest via the p53/p21 pathway
-
Fragkos M, Jurvansuu J, Beard P. H2AX is required for cell cycle arrest via the p53/p21 pathway. Mol Cell Biol 2009; 29:2828-2840.
-
(2009)
Mol Cell Biol
, vol.29
, pp. 2828-2840
-
-
Fragkos, M.1
Jurvansuu, J.2
Beard, P.3
-
17
-
-
30744445418
-
H2AX prevents DNA breaks from progressing to chromosome breaks and translocations
-
Franco S, Gostissa M, Zha S, et al. H2AX prevents DNA breaks from progressing to chromosome breaks and translocations. Mol Cell 2006; 21:201-214.
-
(2006)
Mol Cell
, vol.21
, pp. 201-214
-
-
Franco, S.1
Gostissa, M.2
Zha, S.3
-
18
-
-
0037711771
-
Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaks
-
Celeste A, Fernandez-Capetillo O, Kruhlak MJ, et al. Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaks. Nat Cell Biol 2003; 5:675-679.
-
(2003)
Nat Cell Biol
, vol.5
, pp. 675-679
-
-
Celeste, A.1
Fernandez-Capetillo, O.2
Kruhlak, M.J.3
-
19
-
-
0036903037
-
DNA damage-induced G2-M checkpoint activation by histone H2AX and 53BP1
-
Fernandez-Capetillo O, Chen HT, Celeste A, et al. DNA damage-induced G2-M checkpoint activation by histone H2AX and 53BP1. Nat Cell Biol 2002; 4:993-997.
-
(2002)
Nat Cell Biol
, vol.4
, pp. 993-997
-
-
Fernandez-Capetillo, O.1
Chen, H.T.2
Celeste, A.3
-
20
-
-
79551661127
-
Who's who in human recombination: BRCA2 and RAD52
-
Liu J, Heyer WD. Who's who in human recombination: BRCA2 and RAD52. Proc Natl Acad Sci USA 2011; 108:441-442.
-
(2011)
Proc Natl Acad Sci USA
, vol.108
, pp. 441-442
-
-
Liu, J.1
Heyer, W.D.2
-
21
-
-
2942722673
-
Loss of Rad52 partially rescues tumorigenesis and T-cell maturation in Atm-deficient mice
-
Treuner K, Helton R, Barlow C. Loss of Rad52 partially rescues tumorigenesis and T-cell maturation in Atm-deficient mice. Oncogene 2004; 23:4655-4661.
-
(2004)
Oncogene
, vol.23
, pp. 4655-4661
-
-
Treuner, K.1
Helton, R.2
Barlow, C.3
-
22
-
-
78149425175
-
Regulation of homologous recombination in eukaryotes
-
Heyer WD, Ehmsen KT, Liu J. Regulation of homologous recombination in eukaryotes. Annu Rev Genet 2010; 44:113-139.
-
(2010)
Annu Rev Genet
, vol.44
, pp. 113-139
-
-
Heyer, W.D.1
Ehmsen, K.T.2
Liu, J.3
-
24
-
-
4744345878
-
C-Jun-deficient cells undergo premature senescence as a result of spontaneous DNA damage accumulation
-
MacLaren A, Black EJ, Clark W, Gillespie DA. C-Jun-deficient cells undergo premature senescence as a result of spontaneous DNA damage accumulation. Mol Cell Biol 2004; 24:9006-9018.
-
(2004)
Mol Cell Biol
, vol.24
, pp. 9006-9018
-
-
MacLaren, A.1
Black, E.J.2
Clark, W.3
Gillespie, D.A.4
|