GAD67 deficiency in parvalbumin interneurons produces deficits in inhibitory transmission and network disinhibition in mouse prefrontal cortex

Cerebral Cortex

Volumn 25, Issue 5, 2015, Pages 1290-1296

  Lazarus, Matthew S ^a,b,c   Krishnan, Keerthi ^a   Huang, Z Josh ^a  

^a Simons Center for Quantitative Biology   (United States)

^b STONY BROOK UNIVERSITY   (United States)

^c STONY BROOK UNIVERSITY   (United States)

Author keywords

GABA; GAD67; Neocortex; Parvalbumin

Indexed keywords

4 AMINOBUTYRIC ACID RECEPTOR; GLUTAMATE DECARBOXYLASE 67; PARVALBUMIN; 4 AMINOBUTYRIC ACID; GLUTAMATE DECARBOXYLASE; GLUTAMATE DECARBOXYLASE 1;

ADOLESCENT; ADULT; ANIMAL TISSUE; ARTICLE; BRAIN NERVE CELL; CONTROLLED STUDY; FEMALE; GABAERGIC TRANSMISSION; GAD1 GENE; GENE; INTERNEURON; JUVENILE; MALE; MOUSE; NERVE CELL EXCITABILITY; NERVE CELL NETWORK; NONHUMAN; PREFRONTAL CORTEX; PRIORITY JOURNAL; PYRAMIDAL NERVE CELL; SYNAPTIC INHIBITION; AGING; ANIMAL; DEFICIENCY; ELECTROPHYSIOLOGY; GENETICS; METABOLISM; PHYSIOLOGY; SYNAPTIC TRANSMISSION; TRANSGENIC MOUSE;

AGING; ANIMALS; ELECTROPHYSIOLOGY; FEMALE; GABAERGIC NEURONS; GAMMA-AMINOBUTYRIC ACID; GLUTAMATE DECARBOXYLASE; INTERNEURONS; MALE; MICE; MICE, TRANSGENIC; NERVE NET; PARVALBUMINS; PREFRONTAL CORTEX; PYRAMIDAL CELLS; SYNAPTIC TRANSMISSION;

EID: 84928905835     PISSN: 10473211     EISSN: 14602199     Source Type: Journal    
DOI: 10.1093/cercor/bht322     Document Type: Article

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