Differential effects of buffer pH on Ca2+-induced ROS emission with inhibited mitochondrial complexes I and III

Frontiers in Physiology

Volumn 6, Issue MAR, 2015, Pages

  Lindsay, Daniel P ^a   Camara, Amadou K S ^a   Stowe, David F ^a,b,c   Lubbe, Ryan ^a   Aldakkak, Mohammed ^a  

^a MEDICAL COLLEGE OF WISCONSIN   (United States)

^b CLEMENT J ZABLOCKI VETERANS AFFAIRS MEDICAL CENTER   (United States)

^c Marquette University   (United States)

Author keywords

Ca2+; Mitochondrial complex I; Mitochondrial complex III; Mitochondrial permeability transition pore; pH; Reactive oxygen species; Simulated ischemia

Indexed keywords

ANTIMYCIN A1; BUFFER; CALCIUM CHLORIDE; CALCIUM ION; HYDROGEN PEROXIDE; MITOCHONDRIAL PERMEABILITY TRANSITION PORE; PYRUVIC ACID; REACTIVE OXYGEN METABOLITE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE DEHYDROGENASE (UBIQUINONE); ROTENONE; SUCCINIC ACID; UBIQUINOL CYTOCHROME C REDUCTASE;

ANIMAL CELL; ARTICLE; CONTROLLED STUDY; CYTOSOL; ENZYME INHIBITION; GUINEA PIG; HEART MITOCHONDRION; IN VIVO STUDY; MITOCHONDRION SWELLING; NONHUMAN; PH; REPERFUSION INJURY;

EID: 84926429143     PISSN: None     EISSN: 1664042X     Source Type: Journal    
DOI: 10.3389/fphys.2015.00058     Document Type: Article

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