Inhibition of glycogen synthase kinase 3 ameliorates liver ischemia/reperfusion injury via an energy-dependent mitochondrial mechanism

Journal of Hepatology

Volumn 61, Issue 4, 2014, Pages 816-824

  Fu, Hailong ^a   Xu, Haitao ^a   Chen, Huan ^b   Li, Yonghua ^a   Li, Weiwei ^a   Zhu, Qiufeng ^a   Zhang, Qingqing ^a   Yuan, Hongbin ^a   Liu, Fang ^b   Wang, Quanxing ^b   Miao, Mingyong ^b   Shi, Xueyin ^a  

^a SECOND MILITARY MEDICAL UNIVERSITY   (China)

^b SECOND MILITARY MEDICAL UNIVERSITY   (China)

Author keywords

Energy metabolism; Ischemia reperfusion injury; Mitochondria; Reactive oxygen; Signal transduction

Indexed keywords

2 (2,4 DICHLOROPHENYL) 3 (1 METHYL 3 INDOLYL)MALEIMIDE; ADENOSINE TRIPHOSPHATASE INHIBITOR; ALANINE AMINOTRANSFERASE; ASPARTATE AMINOTRANSFERASE; CATALASE; GLUCOSE; GLYCOGEN SYNTHASE KINASE 3; MITOCHONDRIAL PERMEABILITY TRANSITION PORE; PROTON TRANSPORTING ADENOSINE TRIPHOSPHATE SYNTHASE; REACTIVE OXYGEN METABOLITE; SUPEROXIDE DISMUTASE; INDOLE DERIVATIVE; MALEIMIDE DERIVATIVE; PROTECTIVE AGENT; PROTON TRANSPORTING ADENOSINE TRIPHOSPHATASE;

AGING; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL PROTECTION; CONTROLLED STUDY; DRUG MECHANISM; ENERGY BALANCE; ENZYME INHIBITION; ISCHEMIC PRECONDITIONING; JUVENILE ANIMAL; LIVER ISCHEMIA; LIVER PROTECTION; MALE; METABOLIC REGULATION; MITOCHONDRIAL RESPIRATION; NONHUMAN; PRIORITY JOURNAL; RAT; REPERFUSION INJURY; SENESCENCE; SIGNAL TRANSDUCTION; ANIMAL; ANTAGONISTS AND INHIBITORS; ENERGY METABOLISM; LIVER MITOCHONDRION; METABOLISM; PHYSIOLOGY; PROCEDURES; SPRAGUE DAWLEY RAT;

AGING; ANIMALS; CYTOPROTECTION; ENERGY METABOLISM; GLYCOGEN SYNTHASE KINASE 3; INDOLES; ISCHEMIC PRECONDITIONING; MALEIMIDES; MITOCHONDRIA, LIVER; PROTECTIVE AGENTS; PROTON-TRANSLOCATING ATPASES; RATS; RATS, SPRAGUE-DAWLEY; REPERFUSION INJURY;

EID: 84926415778     PISSN: 01688278     EISSN: 16000641     Source Type: Journal    
DOI: 10.1016/j.jhep.2014.05.017     Document Type: Article

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