Experimental heart failure modelled by the cardiomyocyte-specific loss of an epigenome modifier, DNMT3B

Journal of Molecular and Cellular Cardiology

Volumn 82, Issue , 2015, Pages 174-183

  Vujic, A ^a,b   Robinson, E L ^a   Ito, M ^a   Haider, S ^c   Ackers Johnson, M ^a,b,d   See, K ^d   Methner, C ^a   Figg, N ^a   Brien, P ^e   Roderick, H L ^e   Skepper, J ^a   A Ferguson Smith, ^a   Foo, R S ^a,b,d  

^a UNIVERSITY OF CAMBRIDGE   (United Kingdom)

^b NATIONAL UNIVERSITY HEALTH SYSTEM   (Singapore)

^c QUEEN MARY UNIVERSITY OF LONDON   (United Kingdom)

^d GENOME INSTITUTE OF SINGAPORE   (Singapore)

^e BABRAHAM INSTITUTE   (United Kingdom)

Author keywords

Alternative splicing; DNA methylation; Epigenome; Heart failure

Indexed keywords

DNA METHYLTRANSFERASE; DNA METHYLTRANSFERASE 3B; RNA; DNA (CYTOSINE 5) METHYLTRANSFERASE; MYOSIN HEAVY CHAIN; PROTEIN AGGREGATE; UBIQUITIN;

ADULT ANIMAL; ALTERNATIVE RNA SPLICING; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CELL LOSS; CONTROLLED STUDY; DISEASE COURSE; DISEASE SEVERITY; DNA METHYLATION; EPIGENOME; EXON; EXPERIMENTAL HEART FAILURE; GENE; GENE DELETION; GENOME; HEART DISEASE; HEART HYPERTROPHY; HEART MUSCLE; HEART MUSCLE CELL; HEART MUSCLE FIBROSIS; HUMAN; HUMAN CELL; KNOCKOUT MOUSE; MOUSE; MYH7 GENE; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; SARCOMERE; SYSTOLE; ANIMAL; ANTIBODY SPECIFICITY; DISEASE MODEL; FIBROSIS; GENE EXPRESSION REGULATION; GENETIC EPIGENESIS; GENETICS; HEART FAILURE; METABOLISM; PATHOLOGY; PATHOPHYSIOLOGY; PROTEIN DEGRADATION; SYSTOLIC HEART FAILURE;

MUS;

ALTERNATIVE SPLICING; ANIMALS; DISEASE MODELS, ANIMAL; DNA (CYTOSINE-5-)-METHYLTRANSFERASE; DNA METHYLATION; EPIGENESIS, GENETIC; FIBROSIS; GENE DELETION; GENE EXPRESSION REGULATION; HEART FAILURE; HEART FAILURE, SYSTOLIC; HUMANS; MICE; MICE, KNOCKOUT; MYOCARDIUM; MYOCYTES, CARDIAC; MYOSIN HEAVY CHAINS; ORGAN SPECIFICITY; PROTEIN AGGREGATES; PROTEOLYSIS; SARCOMERES; UBIQUITIN;

EID: 84925432517     PISSN: 00222828     EISSN: 10958584     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2015.03.007     Document Type: Article

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