TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain

Journal of Neuroinflammation

Volumn 11, Issue 1, 2014, Pages

  Wang, Lan Wan ^a,b,d   Chang, Ying Chao ^e   Chen, Shyi Jou ^f   Tseng, Chien Hang ^g   Tu, Yi Fang ^g   Liao, Nan Shih ^h   Huang, Chao Ching ^b,c,g   Ho, Chien Jung ^g  

^a CHI MEI MEDICAL CENTER   (Taiwan)

^b TAIPEI MEDICAL UNIVERSITY   (Taiwan)

^c TAIPEI MEDICAL UNIVERSITY   (Taiwan)

^d CHUNG SHAN MEDICAL UNIVERSITY   (Taiwan)

^e CHANG GUNG UNIVERSITY COLLEGE OF MEDICINE   (Taiwan)

^f TRI SERVICE GENERAL HOSPITAL   (Taiwan)

^g NATIONAL CHENG KUNG UNIVERSITY HOSPITAL   (Taiwan)

^h INSTITUTE OF MOLECULAR BIOLOGY   (Taiwan)

Author keywords

c Jun N terminal kinase; Hypoxic ischemia; Immature brain; Lipopolysaccharide; Neuroinflammation; Neurovascular unit; Tumor necrosis factor receptor 1

Indexed keywords

(1,3 BENZOTHIAZOL 2 YL)[2 [2 (3 PYRIDINYL)ETHYLAMINO] 4 PYRIMIDINYL]ACETONITRILE; CASPASE 3; ETANERCEPT; LIPOPOLYSACCHARIDE; SODIUM CHLORIDE; STRESS ACTIVATED PROTEIN KINASE; TUMOR NECROSIS FACTOR ALPHA; TUMOR NECROSIS FACTOR RECEPTOR 1; TUMOR NECROSIS FACTOR RECEPTOR 2; TNFRSF1A PROTEIN, MOUSE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BLOOD BRAIN BARRIER; BRAIN CORTEX; BRAIN INJURY; BRAIN MATURATION; BRAIN PROTECTION; CAPILLARY ENDOTHELIAL CELL; CELL ACTIVATION; CELLULAR DISTRIBUTION; CEREBROVASCULAR DISEASE; CONTROLLED STUDY; DRUG DOSE COMPARISON; ENZYME ACTIVATION; ENZYME DEGRADATION; ENZYME PHOSPHORYLATION; HYPOXIC ISCHEMIC ENCEPHALOPATHY; IMMUNOBLOTTING; IMMUNOFLUORESCENCE TEST; IMMUNOHISTOCHEMISTRY; KNOCKOUT MOUSE; MICROGLIA; MOUSE; NERVOUS SYSTEM INFLAMMATION; NONHUMAN; OLIGODENDROGLIAL PRECURSOR CELL; PATHOPHYSIOLOGY; PROTEIN ANALYSIS; PROTEIN EXPRESSION; PUERPERIUM; RECEPTOR UPREGULATION; SIGNAL TRANSDUCTION; STEM CELL; UPREGULATION; WHITE MATTER INJURY; WILD TYPE MOUSE; 129 MOUSE; ANIMAL; BIOSYNTHESIS; BRAIN; C57BL MOUSE; CHEMICALLY INDUCED; DRUG EFFECTS; FEMALE; INFLAMMATION; MALE; METABOLISM; NEWBORN; PATHOLOGY; PHYSIOLOGY;

ANIMALS; ANIMALS, NEWBORN; BLOOD-BRAIN BARRIER; BRAIN; FEMALE; HYPOXIA-ISCHEMIA, BRAIN; INFLAMMATION; LIPOPOLYSACCHARIDES; MALE; MAP KINASE SIGNALING SYSTEM; MICE; MICE, 129 STRAIN; MICE, INBRED C57BL; MICE, KNOCKOUT; RECEPTORS, TUMOR NECROSIS FACTOR, TYPE I;

EID: 84924867463     PISSN: None     EISSN: 17422094     Source Type: Journal    
DOI: 10.1186/s12974-014-0215-2     Document Type: Article

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