Prion protein and copper cooperatively protect neurons by modulating NMDA receptor through S-nitrosylation

Antioxidants and Redox Signaling

Volumn 22, Issue 9, 2015, Pages 772-784

  Gasperini, Lisa ^a   Meneghetti, Elisa ^a   Pastore, Beatrice ^a   Benetti, Federico ^a,c   Legname, Giuseppe ^a,b  

^a SISSA   (Italy)

^b SINCROTRONE TRIESTE   (Italy)

^c ECSIN European Center for the Sustainable Impact of Nanotechnology   (Italy)

Author keywords

[No Author keywords available]

Indexed keywords

COPPER; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; N METHYL DEXTRO ASPARTIC ACID RECEPTOR 1; N METHYL DEXTRO ASPARTIC ACID RECEPTOR 2A; NITRIC OXIDE DONOR; NITRIC OXIDE SYNTHASE; PRION PROTEIN; CYSTEINE; NITRIC OXIDE; PRION;

ADULT; ANIMAL TISSUE; ARTICLE; CHELATION; CONTROLLED STUDY; ENZYME INHIBITION; EXCITOTOXICITY; HIPPOCAMPUS; MOUSE; NEUROPROTECTION; NITROSYLATION; NONHUMAN; PRIORITY JOURNAL; PROTEIN FUNCTION; S NITROSYLATION; ANIMAL; CELL CULTURE; CYTOLOGY; GENETICS; METABOLISM; NERVE CELL; PRION; PROTEIN PROCESSING; TRANSGENIC MOUSE;

ANIMALS; CELLS, CULTURED; COPPER; CYSTEINE; HIPPOCAMPUS; MICE; MICE, TRANSGENIC; NEURONS; NITRIC OXIDE; PRIONS; PROTEIN PROCESSING, POST-TRANSLATIONAL; RECEPTORS, N-METHYL-D-ASPARTATE;

EID: 84924701142     PISSN: 15230864     EISSN: 15577716     Source Type: Journal    
DOI: 10.1089/ars.2014.6032     Document Type: Article

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