Hydrogen sulfide attenuates the development of diabetic cardiomyopathy

Clinical Science

Volumn 128, Issue 5, 2015, Pages 325-335

  Zhou, Xiang ^a,b   An, Guoyin ^b   Lu, Xiang ^a  

^a THE SECOND AFFILIATED HOSPITAL OF NANJING MEDICAL UNIVERSITY   (China)

^b THE SECOND AFFILIATED HOSPITAL OF SOOCHOW UNIVERSITY   (China)

Author keywords

Apoptosis; Diabetic cardiomyopathy; Hydrogen sulfide; Inflammation; Oxidative stress

Indexed keywords

2 MORPHOLINO 8 PHENYLCHROMONE; 4 (4 FLUOROPHENYL) 2 (4 METHYLSULFINYLPHENYL) 5 (4 PYRIDYL)IMIDAZOLE; ANTHRA[1,9 CD]PYRAZOL 6(2H) ONE; GLUTATHIONE PEROXIDASE; HEME OXYGENASE 1; HYDROGEN SULFIDE; INTERLEUKIN 1BETA; INTERLEUKIN 6; INTERLEUKIN 8; MALONALDEHYDE; MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE P38; PHOSPHATIDYLINOSITOL 3 KINASE; PROTEIN KINASE B; REACTIVE OXYGEN METABOLITE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE (PHOSPHATE) DEHYDROGENASE (QUINONE); SMALL INTERFERING RNA; SODIUM HYDROSULFIDE; STRESS ACTIVATED PROTEIN KINASE; SUPEROXIDE DISMUTASE; TRANSCRIPTION FACTOR NRF2; TUMOR NECROSIS FACTOR ALPHA; UNCLASSIFIED DRUG; AUTACOID; CYTOKINE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ANTIOXIDANT RESPONSIVE ELEMENT; APOPTOSIS; ARTICLE; CARDIAC MUSCLE; CONTROLLED STUDY; DIABETIC CARDIOMYOPATHY; DRUG BLOOD LEVEL; ECHOCARDIOGRAPHY; GLUCOSE INTAKE; HEART; HEART LEFT VENTRICLE FUNCTION; HEART MUSCLE FIBROSIS; HEART PROTECTION; HEART VENTRICLE HYPERTROPHY; HISTOPATHOLOGY; HYPERGLYCEMIA; INFLAMMATION; MALE; NEWBORN; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; SIGNAL TRANSDUCTION; STREPTOZOTOCIN-INDUCED DIABETES MELLITUS; UPREGULATION; ANIMAL; DIABETIC CARDIOMYOPATHIES; DRUG EFFECTS; ECHOGRAPHY; METABOLISM; PATHOLOGY; SPRAGUE DAWLEY RAT;

ANIMALS; CYTOKINES; DIABETIC CARDIOMYOPATHIES; HYDROGEN SULFIDE; INFLAMMATION MEDIATORS; MALE; OXIDATIVE STRESS; RATS, SPRAGUE-DAWLEY;

EID: 84922392720     PISSN: 01435221     EISSN: None     Source Type: Journal    
DOI: 10.1042/CS20140460     Document Type: Article

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