Decreased myocardial injury and improved contractility after administration of a peptide derived against the alpha-interacting domain of the L-type calcium channel

Journal of the American Heart Association

Volumn 3, Issue 3, 2014, Pages

  Viola, Helena M ^a   Jordan, Maria C ^b   Roos, Kenneth P ^b   Hool, Livia C ^a  

^a UNIVERSITY OF WESTERN AUSTRALIA   (Australia)

^b UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

Ion channels; Ischemia; Peptides; Reperfusion

Indexed keywords

ALPHA INTERACTING DOMAIN TRANSACTIVATOR OF TRANSCRIPTION PEPTIDE; CALCIUM CHANNEL L TYPE; CALCIUM ION; CARDIOVASCULAR AGENT; CREATINE KINASE; FLAVOPROTEIN; FURA 2; LACTATE DEHYDROGENASE; NITROBLUE TETRAZOLIUM; PEPTIDE DERIVATIVE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE; SUPEROXIDE; UNCLASSIFIED DRUG; AID-TAT PEPTIDE; CALCIUM; CARDIOTONIC AGENT; PEPTIDE FRAGMENT;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BIOSYNTHESIS; CELL FUNCTION; CONTROLLED STUDY; DRUG MECHANISM; ENZYME RELEASE; EX VIVO STUDY; GUINEA PIG; HEART INFARCTION SIZE; HEART LEFT VENTRICLE CONTRACTILITY; HEART MITOCHONDRION; HEART MUSCLE INJURY; HEART MUSCLE ISCHEMIA; HEART MUSCLE METABOLISM; MALE; MITOCHONDRIAL MEMBRANE POTENTIAL; MOUSE; NONHUMAN; NUCLEOTIDE METABOLISM; PRIORITY JOURNAL; PROTEIN METABOLISM; PROTEIN TRANSPORT; RAT; REPERFUSION INJURY; ANIMAL; C57BL MOUSE; CHEMISTRY; DISEASE MODEL; DRUG EFFECTS; HEART CONTRACTION; HEART MUSCLE CELL; METABOLISM; MYOCARDIAL INFARCTION; MYOCARDIAL REPERFUSION INJURY; PATHOPHYSIOLOGY; PHYSIOLOGY;

ANIMALS; CALCIUM; CALCIUM CHANNELS, L-TYPE; CARDIOTONIC AGENTS; CREATINE KINASE; DISEASE MODELS, ANIMAL; GUINEA PIGS; L-LACTATE DEHYDROGENASE; MALE; MICE, INBRED C57BL; MITOCHONDRIA, HEART; MYOCARDIAL CONTRACTION; MYOCARDIAL INFARCTION; MYOCARDIAL REPERFUSION INJURY; MYOCYTES, CARDIAC; PEPTIDE FRAGMENTS; RATS; SUPEROXIDES;

EID: 84921800577     PISSN: None     EISSN: 20479980     Source Type: Journal    
DOI: 10.1161/JAHA.114.000961     Document Type: Article

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