Mitochondrial complex i defects increase ubiquitin in substantia nigra

Brain Research

Volumn 1594, Issue , 2015, Pages 82-91

  Song, Lanying ^a   Cortopassi, Gino ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

ComplexI; Dopaminergic neurons; Neurofilaments; Parkinsons disease; Ubiquitin

Indexed keywords

CATECHOL METHYLTRANSFERASE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE DEHYDROGENASE (UBIQUINONE); UBIQUITIN; NDUFS4 PROTEIN, MOUSE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; DISORDERS OF MITOCHONDRIAL FUNCTIONS; DOPAMINE METABOLISM; DOPAMINERGIC NERVE CELL; ENZYME ACTIVITY; ENZYME INHIBITION; FEMALE; HIPPOCAMPUS; IMMUNOHISTOCHEMISTRY; IMMUNOPRECIPITATION; MALE; MASS SPECTROMETRY; MESENCEPHALON; MITOCHONDRIAL COMPLEX I DEFECT; MOUSE; NERVE CELL NECROSIS; NERVE DEGENERATION; NEUROFILAMENT; NONHUMAN; OXIDATIVE STRESS; PARKINSON DISEASE; RESPIRATORY CHAIN; SUBSTANTIA NIGRA; SUBSTANTIA NIGRA PARS COMPACTA; WESTERN BLOTTING; ANIMAL; DEFICIENCY; DISEASE MODEL; INTERMEDIATE FILAMENT; KNOCKOUT MOUSE; METABOLISM; POLYMERASE CHAIN REACTION;

MUS;

ANIMALS; BLOTTING, WESTERN; DISEASE MODELS, ANIMAL; ELECTRON TRANSPORT COMPLEX I; IMMUNOHISTOCHEMISTRY; IMMUNOPRECIPITATION; INTERMEDIATE FILAMENTS; MASS SPECTROMETRY; MICE; MICE, KNOCKOUT; PARKINSON DISEASE; POLYMERASE CHAIN REACTION; SUBSTANTIA NIGRA; UBIQUITIN;

EID: 84919632509     PISSN: 00068993     EISSN: 18726240     Source Type: Journal    
DOI: 10.1016/j.brainres.2014.11.013     Document Type: Article

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