Glucocerebrosidase depletion enhances cell-to-cell transmission of α-synuclein

Nature Communications

Volumn 5, Issue , 2014, Pages

  Bae, Eun Jin ^a   Yang, Na Young ^a   Song, Miyoung ^a   Lee, Cheol Soon ^b   Lee, Jun Sung ^a   Jung, Byung Chul ^a,c   Lee, He Jin ^b   Kim, Seokjoong ^d   Masliah, Eliezer ^e   Sardi, Sergio Pablo ^f   Lee, Seung Jae ^a  

^a Konkuk University   (South Korea)

^b Konkuk University School of Medicine   (South Korea)

^c Yonsei University Wonju   (South Korea)

^d Seoul National University   (South Korea)

^e UNIVERSITY OF CALIFORNIA   (United States)

^f SANOFI   (France)

Author keywords

[No Author keywords available]

Indexed keywords

ALPHA SYNUCLEIN; GLUCOSYLCERAMIDASE; BETA GLUCOSIDASE; GBA2 PROTEIN, HUMAN;

AGGREGATE; CYTOLOGY; ENZYME ACTIVITY; NERVOUS SYSTEM DISORDER;

ARTICLE; CONTROLLED STUDY; DISEASE ASSOCIATION; EXOCYTOSIS; HUMAN; HUMAN CELL; NEUROPATHOLOGY; PARKINSON DISEASE; PROTEIN AGGREGATION; PROTEIN DEPLETION; PROTEIN TRANSPORT; ANIMAL; CELL COMMUNICATION; CELL LINE; GENE INACTIVATION; GENETICS; LYSOSOME; METABOLISM; PATHOLOGY; TRANSGENIC MOUSE;

ALPHA-SYNUCLEIN; ANIMALS; BETA-GLUCOSIDASE; CELL COMMUNICATION; CELL LINE; EXOCYTOSIS; GENE KNOCKOUT TECHNIQUES; HUMANS; LYSOSOMES; MICE, TRANSGENIC; PARKINSON DISEASE; PROTEIN TRANSPORT;

EID: 84907360500     PISSN: None     EISSN: 20411723     Source Type: Journal    
DOI: 10.1038/ncomms5755     Document Type: Article

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