SCOPUS 정보 검색 플랫폼

Volumn 140, Issue , 2014, Pages 143-152

Cu(II) inhibits hIAPP fibrillation and promotes hIAPP-induced beta cell apoptosis through induction of ROS-mediated mitochondrial dysfunction

(5) Ma, Lijuan a Li, Xiaoling a Wang, Yi a Zheng, Wenjie a Chen, Tianfeng a

a JINAN UNIVERSITY (China)

Author keywords

Apoptosis; Copper compound; Fibrosis; hIAPP

Indexed keywords

AMYLIN; AMYLOID; ANION; CATION; COPPER CHLORIDE; COPPER SULFATE; CUPRIC ION; GLYCINATE COPPER; HUMAN ISLET AMYLOID POLYPEPTIDE; REACTIVE OXYGEN METABOLITE; UNCLASSIFIED DRUG; COPPER;

ANIMAL CELL; APOPTOSIS; ARTICLE; ATOMIC FORCE MICROSCOPY; CONTROLLED STUDY; CYTOTOXICITY; DISORDERS OF MITOCHONDRIAL FUNCTIONS; FIBROSIS; IN VITRO STUDY; INS 1 CELL LINE; NON INSULIN DEPENDENT DIABETES MELLITUS; NONHUMAN; PANCREAS ISLET BETA CELL; PARTICLE SIZE; PROTEIN AGGREGATION; PROTEIN INTERACTION; RAT; TRANSMISSION ELECTRON MICROSCOPY; ANIMAL; ANTAGONISTS AND INHIBITORS; CIRCULAR DICHROISM; CYTOLOGY; DRUG EFFECTS; FLUORESCENCE; HUMAN; METABOLISM; MITOCHONDRION; PANCREAS ISLET; PHYSIOLOGY; TUMOR CELL LINE;

ANIMALS; APOPTOSIS; CELL LINE, TUMOR; CIRCULAR DICHROISM; COPPER; FLUORESCENCE; HUMANS; ISLET AMYLOID POLYPEPTIDE; ISLETS OF LANGERHANS; MICROSCOPY, ATOMIC FORCE; MITOCHONDRIA; RATS; REACTIVE OXYGEN SPECIES;

EID: 84906959891 PISSN: 01620134 EISSN: 18733344 Source Type: Journal
DOI: 10.1016/j.jinorgbio.2014.07.002 Document Type: Article

Times cited : (47)

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