-
1
-
-
37549038706
-
Interaction of cardiovascular risk factors with myocardial ischemia/reperfusion injury, preconditioning, and postconditioning
-
doi:10.1124/pr.107.06002
-
Ferdinandy P, Schulz R, Baxter GF (2007) Interaction of cardiovascular risk factors with myocardial ischemia/reperfusion injury, preconditioning, and postconditioning. Pharmacol Rev 59: 418-458. doi:10.1124/pr.107.06002.
-
(2007)
Pharmacol Rev
, vol.59
, pp. 418-458
-
-
Ferdinandy, P.1
Schulz, R.2
Baxter, G.F.3
-
2
-
-
37849023059
-
Cardioprotection by ischemic postconditioning is lost in aged and STAT3-deficient mice
-
doi:10.1161/CIRCRESAHA.107.164699
-
Boengler K, Buechert A, Heinen Y, Roeskes C, Hilfiker-Kleiner D, et al. (2008) Cardioprotection by ischemic postconditioning is lost in aged and STAT3-deficient mice. Circ Res: 102: 131-135. doi:10.1161/CIRCRESAHA.107.164699.
-
(2008)
Circ Res
, vol.102
, pp. 131-135
-
-
Boengler, K.1
Buechert, A.2
Heinen, Y.3
Roeskes, C.4
Hilfiker-Kleiner, D.5
-
3
-
-
56849116269
-
Attenuation of cardioprotective effect by postconditioning in coronary stenosed rat heart and its restoration by carvedilol
-
doi.org/10.1253/circj.CJ-08-0098
-
Oikawa M, Yaoita H, Watanabe K, Maruyama Y (2008) Attenuation of cardioprotective effect by postconditioning in coronary stenosed rat heart and its restoration by carvedilol. Circ J 72: 2081-2086. doi.org/10.1253/circj.CJ- 08-0098.
-
(2008)
Circ J
, vol.72
, pp. 2081-2086
-
-
Oikawa, M.1
Yaoita, H.2
Watanabe, K.3
Maruyama, Y.4
-
4
-
-
4043060632
-
Postconditioning: A form of "modified reperfusion" protects the myocardium by activating the phosphatidylinositol 3-kinase-Akt pathway
-
DOI 10.1161/01.RES.0000138303.76488.fe
-
Tsang A, Hausenloy DJ, Mocanu MM, Yellon DM (2004) Postconditioning: a form of "modified reperfusion" protects the myocardium by activating the phosphatidylinositol 3-kinase-Akt pathway. Circ Res 95: 230-232. (Pubitemid 39056282)
-
(2004)
Circulation Research
, vol.95
, Issue.3
, pp. 230-232
-
-
Tsang, A.1
Hausenloy, D.J.2
Mocanu, M.M.3
Yellon, D.M.4
-
5
-
-
77950350159
-
Effects of a protocol of ischemic postconditioning and/or captopril in hearts of normotensive and hypertensive rats
-
doi:10.1007/s00395-009-0075-6
-
Penna C, Tullio F, Moro F, Folino A, Merlino A, et al. (2010) Effects of a protocol of ischemic postconditioning and/or captopril in hearts of normotensive and hypertensive rats. Basic Res Cardiol 105: 181-192. doi:10.1007/s00395-009-0075-6.
-
(2010)
Basic Res Cardiol
, vol.105
, pp. 181-192
-
-
Penna, C.1
Tullio, F.2
Moro, F.3
Folino, A.4
Merlino, A.5
-
6
-
-
84872295470
-
Cardioprotection by ischemic postconditioning is abrogated in hypertrophied myocardium of spontaneously hypertensive rats
-
doi:10.1097/FJC.0b013e3182760c4d
-
Wagner C, Ebner B, Tillack D, Strasser RH, Weinbrenner C (2013) Cardioprotection by ischemic postconditioning is abrogated in hypertrophied myocardium of spontaneously hypertensive rats. J Cardiovasc Pharmacol 61: 35-41. doi:10.1097/FJC.0b013e3182760c4d.
-
(2013)
J Cardiovasc Pharmacol
, vol.61
, pp. 35-41
-
-
Wagner, C.1
Ebner, B.2
Tillack, D.3
Strasser, R.H.4
Weinbrenner, C.5
-
7
-
-
84877748526
-
Participation of mitochondrial permeability transition pore in the effects of ischemic preconditioning in hypertrophied hearts: Role of NO and mitoK(ATP)
-
doi:10.1016/j.ijcard.2011.10.103
-
Fantinelli JC, Pérez Núñez IA, González Arbeláez LF, Schinella GR, Mosca SM, et al. (2013) Participation of mitochondrial permeability transition pore in the effects of ischemic preconditioning in hypertrophied hearts: Role of NO and mitoK(ATP). Int J Cardiol 166: 173-180. doi:10.1016/j.ijcard.2011.10.103.
-
(2013)
Int J Cardiol
, vol.166
, pp. 173-180
-
-
Fantinelli, J.C.1
Pérez Núñez, I.A.2
González Arbeláez, L.F.3
Schinella, G.R.4
Mosca, S.M.5
-
8
-
-
53249145571
-
The antihypertensive chromogranin a peptide catestatin acts as a novel endocrine/paracrine modulator of cardiac inotropism and lusitropism
-
doi:10.1210/en.2008-0318
-
Angelone T, Quintieri AM, Brar BK, Limchaiyawat PT, Tota B, et al. (2008) The antihypertensive chromogranin a peptide catestatin acts as a novel endocrine/paracrine modulator of cardiac inotropism and lusitropism. Endocrinology 149: 4780-4793. doi:10.1210/en.2008-0318.
-
(2008)
Endocrinology
, vol.149
, pp. 4780-4793
-
-
Angelone, T.1
Quintieri, A.M.2
Brar, B.K.3
Limchaiyawat, P.T.4
Tota, B.5
-
9
-
-
80051960512
-
A novel catestatin-induced antiadrenergic mechanism triggered by the endothelial PI3KeNOS pathway in the myocardium
-
doi:10.1093/cvr/cvr129
-
Bassino E, Fornero S, Gallo MP, Ramella R, Mahata SK, et al. (2011) A novel catestatin-induced antiadrenergic mechanism triggered by the endothelial PI3KeNOS pathway in the myocardium. Cardiovasc Res 91: 617-624. doi:10.1093/cvr/cvr129.
-
(2011)
Cardiovasc Res
, vol.91
, pp. 617-624
-
-
Bassino, E.1
Fornero, S.2
Gallo, M.P.3
Ramella, R.4
Mahata, S.K.5
-
10
-
-
84866705037
-
Chromogranin-A: A multifaceted cardiovascular role in health and disease
-
DOI:10.2174/092986712802430009
-
Angelone T, Mazza R, Cerra MC (2012) Chromogranin-A: a multifaceted cardiovascular role in health and disease. Curr Med Chem 19: 4042-4050. DOI:10.2174/092986712802430009.
-
(2012)
Curr Med Chem
, vol.19
, pp. 4042-4050
-
-
Angelone, T.1
Mazza, R.2
Cerra, M.C.3
-
11
-
-
0036628754
-
Early decline in the catecholamine release-inhibitory peptide catestatin in humans at genetic risk of hypertension
-
DOI 10.1097/00004872-200207000-00020
-
O'Connor DT, Kailasam MT, Kennedy BP, Ziegler MG, Yanaihara N, et al. (2002) Early decline in the catecholamine release-inhibitory peptide catestatin in humans at genetic risk of hypertension. J Hypertens 20: 1335-1345. (Pubitemid 34925678)
-
(2002)
Journal of Hypertension
, vol.20
, Issue.7
, pp. 1335-1345
-
-
O'Connor, D.T.1
Kailasam, M.T.2
Kennedy, B.P.3
Ziegler, M.G.4
Yanaihara, N.5
Parmer, R.J.6
-
12
-
-
84855490818
-
Chromogranin/secretogranin proteins in murine heart: Myocardial production of chromogranin A fragment catestatin (Chga(364-384))
-
doi:10.1007/s00441-010-1059-4
-
Biswas N, Curello E, O'Connor DT, Mahata SK (2010) Chromogranin/ secretogranin proteins in murine heart: myocardial production of chromogranin A fragment catestatin (Chga(364-384)). Cell Tissue Res 342: 353-361. doi:10.1007/s00441-010-1059-4.
-
(2010)
Cell Tissue Res
, vol.342
, pp. 353-361
-
-
Biswas, N.1
Curello, E.2
O'Connor, D.T.3
Mahata, S.K.4
-
13
-
-
84855338182
-
Phosphodiesterase type-2 and NO-dependent S-nitrosylation mediate the cardioinhibition of the antihypertensive catestatin
-
doi:10.1152/ajpheart.00491.2011
-
Angelone T, Quintieri AM, Pasqua T, Gentile S, Tota B, et al. (2012) Phosphodiesterase type-2 and NO-dependent S-nitrosylation mediate the cardioinhibition of the antihypertensive catestatin. Am J Physiol Heart Circ Physiol 302: H431-H442. doi:10.1152/ajpheart.00491.2011.
-
(2012)
Am J Physiol Heart Circ Physiol
, vol.302
-
-
Angelone, T.1
Quintieri, A.M.2
Pasqua, T.3
Gentile, S.4
Tota, B.5
-
14
-
-
78650417675
-
The neuropeptide catestatin acts as a novel angiogenic cytokine via a basic fibroblast growth factor-dependent mechanism
-
doi:10.1161/CIRCRESAHA.110.219493
-
Theurl M, Schgoer W, Albrecht K, Jeschke J, Egger M, et al. (2010) The neuropeptide catestatin acts as a novel angiogenic cytokine via a basic fibroblast growth factor-dependent mechanism. Circ Res 107: 1326-1335. doi:10.1161/CIRCRESAHA.110.219493.
-
(2010)
Circ Res
, vol.107
, pp. 1326-1335
-
-
Theurl, M.1
Schgoer, W.2
Albrecht, K.3
Jeschke, J.4
Egger, M.5
-
15
-
-
79151472633
-
Catestatin improves post-ischemic left ventricular function and decreases ischemia/reperfusion injury in heart
-
doi:10.1007/s10571-010-9598-5
-
Penna C, Alloatti G, Gallo MP, Cerra MC, Levi R, et al. (2010) Catestatin improves post-ischemic left ventricular function and decreases ischemia/reperfusion injury in heart. Cell Mol Neurobiol 30: 1171-1179. doi:10.1007/s10571-010-9598-5.
-
(2010)
Cell Mol Neurobiol
, vol.30
, pp. 1171-1179
-
-
Penna, C.1
Alloatti, G.2
Gallo, M.P.3
Cerra, M.C.4
Levi, R.5
-
16
-
-
84879842838
-
Catestatin reduces myocardial ischaemia/reperfusion injury: Involvement of PI3K/Akt, PKCs, mitochondrial K(ATP) channels and ROS signalling
-
doi:10.1007/s00424-013-1217-0
-
Perrelli MG, Tullio F, Angotti C, Cerra MC, Angelone T, et al. (2013) Catestatin reduces myocardial ischaemia/reperfusion injury: involvement of PI3K/Akt, PKCs, mitochondrial K(ATP) channels and ROS signalling. Pflugers Arch 465: 1031-1040. doi:10.1007/s00424-013-1217-0.
-
(2013)
Pflugers Arch
, vol.465
, pp. 1031-1040
-
-
Perrelli, M.G.1
Tullio, F.2
Angotti, C.3
Cerra, M.C.4
Angelone, T.5
-
17
-
-
80051547653
-
Postischemic early acidosis in cardiac postconditioning modifies the activity of antioxidant enzymes, reduces nitration, and favors protein S-nitrosylation
-
doi:10.1007/s00424-011-0970-1
-
Penna C, Perrelli MG, Tullio F, Moro F, Parisella ML, et al. (2011) Postischemic early acidosis in cardiac postconditioning modifies the activity of antioxidant enzymes, reduces nitration, and favors protein S-nitrosylation. Pflugers Arch. 462: 219-233. doi:10.1007/s00424-011-0970-1.
-
(2011)
Pflugers Arch
, vol.462
, pp. 219-233
-
-
Penna, C.1
Perrelli, M.G.2
Tullio, F.3
Moro, F.4
Parisella, M.L.5
-
18
-
-
84856945096
-
S-nitrosylation: A radical way to protect the heart
-
doi:10.1016/j.yjmcc.2011.08.021
-
Murphy E, Kohr M, Sun J, Nguyen T, Steenbergen C (2012) S-nitrosylation: a radical way to protect the heart. J Mol Cell Cardiol 52: 568-577. doi:10.1016/j.yjmcc.2011.08.021.
-
(2012)
J Mol Cell Cardiol
, vol.52
, pp. 568-577
-
-
Murphy, E.1
Kohr, M.2
Sun, J.3
Nguyen, T.4
Steenbergen, C.5
-
19
-
-
79551656099
-
Cardioprotective pathways during reperfusion: Focus on redox signaling and other modalities of cell signaling
-
doi:10.1089/ars.2010.3245
-
Pagliaro P, Moro F, Tullio F, Perrelli MG, Penna C (2011) Cardioprotective pathways during reperfusion: focus on redox signaling and other modalities of cell signaling. Antioxid Redox Signal 14: 833-850. doi:10.1089/ars.2010.3245.
-
(2011)
Antioxid Redox Signal
, vol.14
, pp. 833-850
-
-
Pagliaro, P.1
Moro, F.2
Tullio, F.3
Perrelli, M.G.4
Penna, C.5
-
20
-
-
80054883388
-
Mitochondrial cyclophilin-D as a potential therapeutic target for postmyocardial infarction heart failure
-
doi:10.1111/j.1582-4934.2010.01235.x
-
Lim SY, Hausenloy DJ, Arjun S, Price AN, Davidson SM, et al. (2011) Mitochondrial cyclophilin-D as a potential therapeutic target for postmyocardial infarction heart failure. J Cell Mol Med 15: 2443-2451. doi:10.1111/j.1582- 4934.2010.01235.x.
-
(2011)
J Cell Mol Med
, vol.15
, pp. 2443-2451
-
-
Lim, S.Y.1
Hausenloy, D.J.2
Arjun, S.3
Price, A.N.4
Davidson, S.M.5
-
21
-
-
23644439061
-
Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to heart failure
-
DOI 10.1172/JCI24682
-
Shiojima I, Sato K, Izumiya Y, Schiekofer S, Ito M, et al. (2005) Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to heart failure. J Clin Invest 115: 2108-2118. doi:10.1172/JCI24682. (Pubitemid 41134152)
-
(2005)
Journal of Clinical Investigation
, vol.115
, Issue.8
, pp. 2108-2118
-
-
Shiojima, I.1
Sato, K.2
Izumiya, Y.3
Schiekofer, S.4
Ito, M.5
Liao, R.6
Colucci, W.S.7
Walsh, K.8
-
22
-
-
84865610131
-
Hypoxia-inducible factor as a therapeutic target for cardioprotection
-
doi:10.1016/j.pharmthera.2012.07.005
-
Ong SG, Hausenloy DJ (2012) Hypoxia-inducible factor as a therapeutic target for cardioprotection. Pharmacol Ther 136: 69-81. doi:10.1016/j. pharmthera.2012.07.005.
-
(2012)
Pharmacol Ther
, vol.136
, pp. 69-81
-
-
Ong, S.G.1
Hausenloy, D.J.2
-
23
-
-
0016208708
-
Cardiac hypertrophy in spontaneously hypertensive rats
-
doi:10.1161/01.RES.35.5.775
-
Sen S, Tarazi RC, Khairallah PA, Bumpus FM (1974) Cardiac hypertrophy in spontaneously hypertensive rats. Circ Res 35: 775-781. doi:10.1161/01.RES.35.5. 775.
-
(1974)
Circ Res
, vol.35
, pp. 775-781
-
-
Sen, S.1
Tarazi, R.C.2
Khairallah, P.A.3
Bumpus, F.M.4
-
24
-
-
84867717542
-
HIF-1a and paradoxical phenomena in cardioprotection
-
doi:10.1093/cvr/cvs145
-
Heusch G (2012) HIF-1a and paradoxical phenomena in cardioprotection. Cardiovasc Res 96: 214-215. doi:10.1093/cvr/cvs145.
-
(2012)
Cardiovasc Res
, vol.96
, pp. 214-215
-
-
Heusch, G.1
-
25
-
-
84881244793
-
GPER mediates cardiotropic effects in spontaneously hypertensive rat hearts
-
doi:10.1371/journal.pone.0069322
-
De Francesco EM, Angelone T, Pasqua T, Pupo M, Cerra MC, et al. (2013) GPER mediates cardiotropic effects in spontaneously hypertensive rat hearts. PLoSOne 8: e69322. doi:10.1371/journal.pone.0069322.
-
(2013)
PLoSOne
, vol.8
-
-
De Francesco, E.M.1
Angelone, T.2
Pasqua, T.3
Pupo, M.4
Cerra, M.C.5
-
26
-
-
12244282420
-
Nitroxyl affords thiol-sensitive myocardial protective effects akin to early preconditioning
-
DOI 10.1016/S0891-5849(02)01179-6, PII S0891584902011796
-
Pagliaro P, Mancardi D, Rastaldo R, Penna C, Gattullo D, et al. (2003) Nitroxyl affords thiol-sensitive myocardial protective effects akin to early preconditioning. Free Radic Biol Med 34: 33-43. doi.org/10.1016/S0891-5849(02) 01179-6. (Pubitemid 36043884)
-
(2003)
Free Radical Biology and Medicine
, vol.34
, Issue.1
, pp. 33-43
-
-
Pagliaro, P.1
Mancardi, D.2
Rastaldo, R.3
Penna, C.4
Gattullo, D.5
Miranda, K.M.6
Feelisch, M.7
Wink, D.A.8
Kass, D.A.9
Paolocci, N.10
-
27
-
-
18044380974
-
Platelet-activating factor induces cardioprotection in isolated rat heart akin to ischemic preconditioning: Role of phosphoinositide 3-kinase and protein kinase C activation
-
DOI 10.1152/ajpheart.00599.2004
-
Penna C, Alloatti G, Cappello S, Gattullo D, Berta G, et al. (2005) Plateletactivating factor induces cardioprotection in isolated rat heart akin to ischaemic preconditioning: role of phosphoinositide 3-kinase and protein kinase C activation. Am J Physiol Heart Circ Physiol 288: H2512-H2520. DOI:10.1152/ajpheart.00599.2004. (Pubitemid 40604141)
-
(2005)
American Journal of Physiology - Heart and Circulatory Physiology
, vol.288
, Issue.5
-
-
Penna, C.1
Alloatti, G.2
Cappello, S.3
Gattullo, D.4
Berta, G.5
Mognetti, B.6
Losano, G.7
Pagliaro, P.8
-
28
-
-
84865091899
-
Postconditioning with glucagon like peptide-2 reduces ischemia/reperfusion injury in isolated rat hearts: Role of survival kinases and mitochondrial KATP channels
-
doi:10.1007/s00395-012-0272-6
-
Penna C, Pasqua T, Perrelli MG, Pagliaro P, Cerra MC, et al. (2012) Postconditioning with glucagon like peptide-2 reduces ischemia/reperfusion injury in isolated rat hearts: role of survival kinases and mitochondrial KATP channels. Basic Res Cardiol 107: 272. doi:10.1007/s00395-012-0272-6.
-
(2012)
Basic Res Cardiol
, vol.107
, pp. 272
-
-
Penna, C.1
Pasqua, T.2
Perrelli, M.G.3
Pagliaro, P.4
Cerra, M.C.5
-
29
-
-
77958074379
-
Postconditioning effect of granulocyte colony-stimulating factor is mediated through activation of risk pathway and opening of the mitochondrial KATP channels
-
doi:10.1152/ajpheart.00116.2010
-
Sumi S, Kobayashi H, Yasuda S, Iwasa M, Yamaki T, et al. (2010) Postconditioning effect of granulocyte colony-stimulating factor is mediated through activation of risk pathway and opening of the mitochondrial KATP channels. Am J Physiol Heart Circ Physiol 299: H1174-H1182. doi:10.1152/ ajpheart.00116.2010.
-
(2010)
Am J Physiol Heart Circ Physiol
, vol.299
-
-
Sumi, S.1
Kobayashi, H.2
Yasuda, S.3
Iwasa, M.4
Yamaki, T.5
-
30
-
-
84865337214
-
The role of nitric oxide, reactive oxygen species, and protein kinase C in oxytocin-induced cardioprotection in ischemic rat heart
-
doi:10.1016/j.peptides.2012.08.001
-
Faghihi M, Alizadeh AM, Khori V, Latifpour M, Khodayari S (2012) The role of nitric oxide, reactive oxygen species, and protein kinase C in oxytocin-induced cardioprotection in ischemic rat heart. Peptides 37: 314-319. doi:10.1016/j.peptides.2012.08.001.
-
(2012)
Peptides
, vol.37
, pp. 314-319
-
-
Faghihi, M.1
Alizadeh, A.M.2
Khori, V.3
Latifpour, M.4
Khodayari, S.5
-
31
-
-
73949152865
-
Apoptosis repressor with caspase recruitment domain is dramatically reduced in cardiac, skeletal, and vascular smooth muscle during hypertension
-
doi:10.1016/j.bbrc.2009.12.084
-
Quadrilatero J, Bloemberg D (2010) Apoptosis repressor with caspase recruitment domain is dramatically reduced in cardiac, skeletal, and vascular smooth muscle during hypertension. Biochem Biophys Res Commun 391: 1437-1442. doi:10.1016/j.bbrc.2009.12.084.
-
(2010)
Biochem Biophys Res Commun
, vol.391
, pp. 1437-1442
-
-
Quadrilatero, J.1
Bloemberg, D.2
-
32
-
-
84877052854
-
Nitric oxide synthase-dependent "On/Off" switch and apoptosis in freshwater and aestivating lungfish, Protopterus annectens: Skeletal muscle versus cardiac muscle
-
doi:10.1016/j.niox.2013.03.005
-
Amelio D, Garofalo F, Wong WP, Chew SF, Ip YK, et al. (2013) Nitric oxide synthase-dependent "On/Off" switch and apoptosis in freshwater and aestivating lungfish, Protopterus annectens: Skeletal muscle versus cardiac muscle. Nitric Oxide 32: 1-12. doi:10.1016/j.niox.2013.03.005.
-
(2013)
Nitric Oxide
, vol.32
, pp. 1-12
-
-
Amelio, D.1
Garofalo, F.2
Wong, W.P.3
Chew, S.F.4
Ip, Y.K.5
-
33
-
-
58849106019
-
The cardioprotective effect of postconditioning is mediated by ARC through inhibiting mitochondrial apoptotic pathway
-
doi:10.1007/s10495-008-0296-4
-
Li Y, Ge X, Liu X (2009) The cardioprotective effect of postconditioning is mediated by ARC through inhibiting mitochondrial apoptotic pathway. Apoptosis 14: 164-172. doi:10.1007/s10495-008-0296-4.
-
(2009)
Apoptosis
, vol.14
, pp. 164-172
-
-
Li, Y.1
Ge, X.2
Liu, X.3
-
34
-
-
67649274371
-
Postconditioning induces an anti-apoptotic effect and preserves mitochondrial integrity in isolated rat hearts
-
doi:10.1016/j.bbabio.2009.03.013
-
Penna C, Perrelli MG, Raimondo S, Tullio F, Merlino A, et al. (2009) Postconditioning induces an anti-apoptotic effect and preserves mitochondrial integrity in isolated rat hearts. Biochim Biophys Acta 1787: 794-801. doi:10.1016/j.bbabio.2009.03.013.
-
(2009)
Biochim Biophys Acta
, vol.1787
, pp. 794-801
-
-
Penna, C.1
Perrelli, M.G.2
Raimondo, S.3
Tullio, F.4
Merlino, A.5
-
35
-
-
72449173802
-
Attenuation of myocardial injury by postconditioning: Role of hypoxia inducible factor-1alpha
-
doi:10.1007/s00395-009-0044-0
-
Zhao HX, Wang XL, Wang YH, Wu Y, Li XY, et al. (2010) Attenuation of myocardial injury by postconditioning: role of hypoxia inducible factor-1alpha. Basic Res Cardiol 105: 109-118. doi:10.1007/s00395-009-0044-0.
-
(2010)
Basic Res Cardiol
, vol.105
, pp. 109-118
-
-
Zhao, H.X.1
Wang, X.L.2
Wang, Y.H.3
Wu, Y.4
Li, X.Y.5
-
36
-
-
0036813511
-
Nitric oxide and cardioprotection during ischemia-reperfusion
-
Jugdutt BI (2002) Nitric oxide and cardioprotection during ischemia-reperfusion. Heart Fail Rev 7: 391-405.
-
(2002)
Heart Fail Rev
, vol.7
, pp. 391-405
-
-
Jugdutt, B.I.1
-
37
-
-
37449029816
-
Differential NOS expression in freshwater and aestivating Protopterus dolloi (lungfish): Heart vs kidney readjustments
-
doi.org/10.1016/j.niox.2007.10.004
-
Amelio D, Garofalo F, Brunelli E, Loong AM, Wong WP, et al. (2008) Differential NOS expression in freshwater and aestivating Protopterus dolloi (lungfish): heart vs kidney readjustments. Nitric Oxide 18: 1-10. doi.org/10.1016/j.niox.2007.10.004.
-
(2008)
Nitric Oxide
, vol.18
, pp. 1-10
-
-
Amelio, D.1
Garofalo, F.2
Brunelli, E.3
Loong, A.M.4
Wong, W.P.5
-
38
-
-
0017184389
-
A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding
-
Bradford MM (1976) A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. Anal Biochem 7: 248-254.
-
(1976)
Anal Biochem
, vol.7
, pp. 248-254
-
-
Bradford, M.M.1
-
39
-
-
33646813330
-
2+ channel alpha1 subunit and reduced ischemia/reperfusion injury
-
doi:10.1161/01.RES.0000202707.79018.0a
-
2+ channel alpha1 subunit and reduced ischemia/reperfusion injury. Circ Res 98: 403-411. doi:10.1161/01.RES.0000202707.79018.0a.
-
(2006)
Circ Res
, vol.98
, pp. 403-411
-
-
Sun, J.1
Picht, E.2
Ginsburg, K.S.3
Bers, D.M.4
Steenbergen, C.5
-
40
-
-
0033768870
-
Primary structure and function of the catecholamine release inhibitory peptide catestatin (chromogranin A344-364): Identification of amino acid residues crucial for activity
-
Mahata SK, Mahata M, Wakade AR, O'Connor DT (2000) Primary structure and function of the catecholamine release inhibitory peptide catestatin (chromogranin A344-364): Identification of amino acid residues crucial for activity. Mol Endocrinol 14: 1525-1535.
-
(2000)
Mol Endocrinol
, vol.14
, pp. 1525-1535
-
-
Mahata, S.K.1
Mahata, M.2
Wakade, A.R.3
O'Connor, D.T.4
-
41
-
-
84858266346
-
Cinaciguat, a novel activator of soluble guanylate cyclase, protects against ischemia/reperfusion injury: Role of hydrogen sulfide
-
doi:10.1152/ajpheart.00544.2011
-
Salloum FN, Das A, Samidurai A, Hoke NN, Chau VQ, et al. (2012) Cinaciguat, a novel activator of soluble guanylate cyclase, protects against ischemia/reperfusion injury: role of hydrogen sulfide. Am J Physiol Heart Circ Physiol 302: H1347-H1354. doi:10.1152/ajpheart.00544.2011.
-
(2012)
Am J Physiol Heart Circ Physiol
, vol.302
-
-
Salloum, F.N.1
Das, A.2
Samidurai, A.3
Hoke, N.N.4
Chau, V.Q.5
-
42
-
-
20444402649
-
Postconditioning reduces infarct size via adenosine receptor activation by endogenous adenosine
-
doi:10.1016/j.cardiores. 2005.02.015
-
Kin H, Zatta AJ, Lofye MT, Amerson BS, Halkos ME, et al. (2005) Postconditioning reduces infarct size via adenosine receptor activation by endogenous adenosine. Cardiovasc Res 67: 124-133. doi:10.1016/j.cardiores. 2005.02.015.
-
(2005)
Cardiovasc Res
, vol.67
, pp. 124-133
-
-
Kin, H.1
Zatta, A.J.2
Lofye, M.T.3
Amerson, B.S.4
Halkos, M.E.5
-
43
-
-
79955536647
-
Ischemia/reperfusion injury is increased and cardioprotection by a postconditioning protocol is lost as cardiac hypertrophy develops in nandrolone treated rats
-
doi:10.1007/s00395-010-0143-y
-
Penna C, Tullio F, Perrelli MG, Moro F, Abbadessa G, et al. (2011) Ischemia/reperfusion injury is increased and cardioprotection by a postconditioning protocol is lost as cardiac hypertrophy develops in nandrolone treated rats. Basic Res Cardiol 106: 409-420. doi:10.1007/s00395-010-0143-y.
-
(2011)
Basic Res Cardiol
, vol.106
, pp. 409-420
-
-
Penna, C.1
Tullio, F.2
Perrelli, M.G.3
Moro, F.4
Abbadessa, G.5
-
44
-
-
84856746125
-
Chronic Akt activation accentuates aging-induced cardiac hypertrophy and myocardial contractile dysfunction: Role of autophagy
-
doi:10.1007/s00395-011-0222-8
-
Hua Y, Zhang Y, Ceylan-Isik AF, Wold LE, Nunn JM, et al. (2011) Chronic Akt activation accentuates aging-induced cardiac hypertrophy and myocardial contractile dysfunction: role of autophagy. Basic Res Cardiol. 106: 1173-1191. doi:10.1007/s00395-011-0222-8.
-
(2011)
Basic Res Cardiol
, vol.106
, pp. 1173-1191
-
-
Hua, Y.1
Zhang, Y.2
Ceylan-Isik, A.F.3
Wold, L.E.4
Nunn, J.M.5
-
45
-
-
67749129256
-
Selective activation of PI3Kalpha/Akt/GSK-3beta signalling and cardiac compensatory hypertrophy during recovery from heart failure
-
doi:10.1093/eurjhf/hfp094
-
Braz JC, Gill RM, Corbly AK, Jones BD, Jin N, et al. (2009) Selective activation of PI3Kalpha/Akt/GSK-3beta signalling and cardiac compensatory hypertrophy during recovery from heart failure. Eur J Heart Fail 11: 739-748. doi:10.1093/eurjhf/hfp094.
-
(2009)
Eur J Heart Fail
, vol.11
, pp. 739-748
-
-
Braz, J.C.1
Gill, R.M.2
Corbly, A.K.3
Jones, B.D.4
Jin, N.5
-
46
-
-
84862582485
-
Early apoptosis in different models of cardiac hypertrophy induced by high renin-angiotensin system activity involves CaMKII
-
doi:10.1152/japplphysiol.01383.2011
-
Velez Rueda JO, Palomeque J, Mattiazzi A (2012) Early apoptosis in different models of cardiac hypertrophy induced by high renin-angiotensin system activity involves CaMKII. J Appl Physiol 112: 2110-2120. doi:10.1152/ japplphysiol.01383.2011.
-
(2012)
J Appl Physiol
, vol.112
, pp. 2110-2120
-
-
Velez Rueda, J.O.1
Palomeque, J.2
Mattiazzi, A.3
-
47
-
-
67650245365
-
Regulation and pharmacology of the mitochondrial permeability transition pore
-
doi:10.1093/cvr/cvp151
-
Zorov DB, Juhaszova M, Yaniv Y, Nuss HB, Wang S, et al. (2009) Regulation and pharmacology of the mitochondrial permeability transition pore. Cardiovasc Res 83: 213-225. doi:10.1093/cvr/cvp151.
-
(2009)
Cardiovasc Res
, vol.83
, pp. 213-225
-
-
Zorov, D.B.1
Juhaszova, M.2
Yaniv, Y.3
Nuss, H.B.4
Wang, S.5
-
48
-
-
0032574745
-
ARC, an inhibitor of apoptosis expressed in skeletal muscle and heart that interacts selectively with caspases
-
Koseki T, Inohara N, Chen S, Núñez G (1998) ARC, an inhibitor of apoptosis expressed in skeletal muscle and heart that interacts selectively with caspases. Proc Natl Acad Sci USA 95: 5156-5560.
-
(1998)
Proc Natl Acad Sci USA
, vol.95
, pp. 5156-5560
-
-
Koseki, T.1
Inohara, N.2
Chen, S.3
Núñez, G.4
-
49
-
-
72449173802
-
Attenuation of myocardial injury by postconditioning: Role of hypoxia inducible factor-1alpha
-
doi:10.1007/s00395-009-0044-0
-
Zhao HX, Wang XL, Wang YH, Wu Y, Li XY, et al. (2010) Attenuation of myocardial injury by postconditioning: role of hypoxia inducible factor-1alpha. Basic Res Cardiol 105: 109-118. doi:10.1007/s00395-009-0044-0.
-
(2010)
Basic Res Cardiol
, vol.105
, pp. 109-118
-
-
Zhao, H.X.1
Wang, X.L.2
Wang, Y.H.3
Wu, Y.4
Li, X.Y.5
-
50
-
-
84883158442
-
Full-length human Chromogranin-A cardioactivity: Myocardial, coronary and stimulusinduced processing evidence in normotensive and hypertensive male rat hearts
-
doi:10.1210/en.2012-2210
-
Pasqua T, Corti A, Gentile S, Pochini L, Bianco M, et al. (2013) Full-length human Chromogranin-A cardioactivity: myocardial, coronary and stimulusinduced processing evidence in normotensive and hypertensive male rat hearts. Endocrinology 154: 3353-3365. doi:10.1210/en.2012-2210. Ricerca
-
(2013)
Endocrinology
, vol.154
, pp. 3353-3365
-
-
Pasqua, T.1
Corti, A.2
Gentile, S.3
Pochini, L.4
Bianco, M.5
|