Interleukin-23 mediates the intestinal response to microbial β-1,3-glucan and the development of spondyloarthritis pathology in SKG mice

Arthritis and Rheumatology

Volumn 66, Issue 7, 2014, Pages 1755-1767

  Benham, Helen ^a   Rehaume, Linda M ^a   Hasnain, Sumaira Z ^a   Velasco, Jared ^a   Baillet, Athan C ^a   Ruutu, Merja ^a   Kikly, Kristine ^b   Wang, Ran ^a   Tseng, Hsu Wen ^a   Thomas, Gethin P ^a   Brown, Matthew A ^a   Strutton, Geoffrey ^c   McGuckin, Michael A ^a   Thomas, Ranjeny ^a  

^a UNIVERSITY OF QUEENSLAND   (Australia)

^b ELI LILLY AND COMPANY   (United States)

^c PRINCESS ALEXANDRA HOSPITAL   (Australia)

Author keywords

[No Author keywords available]

Indexed keywords

BETA 1,3 GLUCAN; INTERLEUKIN 17; INTERLEUKIN 22; INTERLEUKIN 23; PROTEIN KINASE ZAP 70;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTHRITIS; ARTICLE; CYTOKINE PRODUCTION; ENDOPLASMIC RETICULUM STRESS; ENTHESITIS; GOBLET CELL; ILEITIS; ILEUM; LAMINA PROPRIA; MONONUCLEAR CELL; MOUSE; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; SPONDYLARTHRITIS;

ANIMALS; ANTIBODIES; BETA-GLUCANS; DISEASE MODELS, ANIMAL; ENDOPLASMIC RETICULUM STRESS; FEMALE; ILEITIS; IMMUNE TOLERANCE; INTERLEUKIN-17; INTERLEUKIN-23 SUBUNIT P19; INTERLEUKINS; INTESTINES; MALE; MICE; MICE, INBRED BALB C; MICE, KNOCKOUT; RECEPTORS, INTERLEUKIN; SPONDYLARTHRITIS;

EID: 84903473100     PISSN: 23265191     EISSN: 23265205     Source Type: Journal    
DOI: 10.1002/art.38638     Document Type: Article

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