Oxidative post-translational modifications develop LONP1 dysfunction in pressure overload heart failure

Circulation: Heart Failure

Volumn 7, Issue 3, 2014, Pages 500-509

  Hoshino, Atsushi ^a   Okawa, Yoshifumi ^a   Ariyoshi, Makoto ^a   Kaimoto, Satoshi ^a   Uchihashi, Motoki ^a   Fukai, Kuniyoshi ^a   Iwai Kanai, Eri ^a,b,c   Matoba, Satoaki ^a  

^a KYOTO PREFECTURAL UNIVERSITY OF MEDICINE   (Japan)

^b MEIJI UNIVERSITY OF INTEGRATIVE MEDICINE   (Japan)

^c TENRI HEALTH CARE UNIVERSITY   (Japan)

Author keywords

Heart failure; Mitochondria; Post translational; Protein processing

Indexed keywords

CATALASE; CYSTEINE; ENDOPEPTIDASE LA; SUPEROXIDE DISMUTASE; TYROSINE;

ARTICLE; DISORDERS OF MITOCHONDRIAL FUNCTIONS; ENZYME ACTIVITY; HEART FAILURE; HEART FUNCTION; HEART MITOCHONDRION; HEART MUSCLE CONTRACTILITY; MITOCHONDRIAL RESPIRATION; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN CARBONYLATION; PROTEIN DEGRADATION; PROTEIN EXPRESSION; PROTEIN HOMEOSTASIS; PROTEIN METABOLISM; PROTEIN PROCESSING; RESPIRATORY CHAIN;

HEART FAILURE; MITOCHONDRIA; PROTEIN PROCESSING, POST-TRANSLATIONAL;

ANIMALS; CYSTEINE; DISEASE MODELS, ANIMAL; HEART FAILURE; MICE; MICE, INBRED C57BL; MITOCHONDRIA, HEART; ORGANOPHOSPHORUS COMPOUNDS; OXIDATION-REDUCTION; PIPERIDINES; PROTEASE LA; PROTEIN PROCESSING, POST-TRANSLATIONAL; RNA, MESSENGER;

EID: 84902782383     PISSN: 19413289     EISSN: 19413297     Source Type: Journal    
DOI: 10.1161/CIRCHEARTFAILURE.113.001062     Document Type: Article

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