Decreased cardiac excitability secondary to reduction of sodium current may be a significant contributor to reduced contractility in a rat model of sepsis

Critical Care

Volumn 18, Issue 2, 2014, Pages

  Koesters, Andrew ^a   Engisch, Kathrin L ^a   Rich, Mark M ^a  

^a WRIGHT STATE UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CALCIUM; NIFEDIPINE; SODIUM; TETRODOTOXIN; SODIUM CHANNEL; SODIUM CHANNEL BLOCKING AGENT;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CALCIUM CURRENT; CELL DAMAGE; CONTROLLED STUDY; EX VIVO STUDY; EXPERIMENTAL LIGATION; FEMALE; HEART FAILURE; HEART MUSCLE CELL; HEART MUSCLE CONTRACTILITY; HEART MUSCLE EXCITABILITY; HEART MUSCLE POTENTIAL; HEART PAPILLARY MUSCLE; MICROELECTRODE; NONHUMAN; PRIORITY JOURNAL; RAT; SEPSIS; SODIUM CURRENT; ACTION POTENTIAL; ANIMAL; CARDIAC MUSCLE CELL; DISEASE MODEL; DRUG EFFECTS; HEART CONTRACTION; ORGAN CULTURE TECHNIQUE; PATHOPHYSIOLOGY; PHYSIOLOGY; WISTAR RAT;

ACTION POTENTIALS; ANIMALS; DISEASE MODELS, ANIMAL; FEMALE; MYOCARDIAL CONTRACTION; MYOCYTES, CARDIAC; ORGAN CULTURE TECHNIQUES; RATS; RATS, WISTAR; SEPSIS; SODIUM CHANNEL BLOCKERS; SODIUM CHANNELS;

EID: 84899639985     PISSN: 13648535     EISSN: 1466609X     Source Type: Journal    
DOI: 10.1186/cc13800     Document Type: Article

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