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Volumn 63, Issue SUPPL., 2014, Pages

Recent progress in the genetics of spontaneously hypertensive rats

Author keywords

Gene targeting; Intermediary phenotypes; Recombinant inbred strains; Spontaneously hypertensive rat; Transgenic

Indexed keywords

ANIMAL; CLASSIFICATION; GENETICS; HUMAN; INSULIN RESISTANCE; METABOLIC SYNDROME X; QUANTITATIVE TRAIT LOCUS; RAT; REVIEW; SPECIES DIFFERENCE; SPONTANEOUSLY HYPERTENSIVE RAT; TRANSGENIC RAT;

EID: 84897594611     PISSN: 08628408     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Review
Times cited : (45)

References (39)
  • 1
    • 68949117500 scopus 로고    scopus 로고
    • Return of the rat
    • ABBOTT A. Return of the rat. Nature 460. 788, 2009.
    • (2009) Nature , vol.460 , pp. 788
    • Abbott, A.1
  • 7
    • 0035968175 scopus 로고    scopus 로고
    • Defective fatty acid uptake in the spontaneously hypertensive rat is a primary determinant of altered glucose metabolism, hyperinsulinemia, and myocardial hypertrophy
    • HAJRI T, IBRAHIMI A, COBURN CT, KNAPP FF JR, KURTZ T, PRAVENEC M, ABUMRAD NA. Defective fatty acid uptake in the spontaneously hypertensive rat is a primary determinant of altered glucose metabolism, hyperinsulinemia, and myocardial hypertrophy. J Biol Chem 276. 23661-23666, 2001.
    • (2001) J Biol Chem , vol.276 , pp. 23661-23666
    • Hajri, T.1    Ibrahimi, A.2    Coburn, C.T.3    Knapp Jr, F.F.4    Kurtz, T.5    Pravenec, M.6    Abumrad, N.A.7
  • 8
    • 84859522892 scopus 로고    scopus 로고
    • Three reasons to abandon low-density lipoprotein targets An open letter to the Adult Treatment Panel IV of the National Institutes of Health
    • HAYWARD RA, KRUMHOLZ HM. Three reasons to abandon low-density lipoprotein targets. An open letter to the Adult Treatment Panel IV of the National Institutes of Health. Circ Cardiovasc Qual Outcomes 5. 2-5, 2012.
    • (2012) Circ Cardiovasc Qual Outcomes , vol.5 , pp. 2-5
    • Hayward, R.A.1    Krumholz, H.M.2
  • 15
    • 78650419995 scopus 로고    scopus 로고
    • Genome-wide association studies will unlock the genetic basis of hypertension. con side of the argument
    • KURTZ TW. Genome-wide association studies will unlock the genetic basis of hypertension. con side of the argument. Hypertension 56. 1021-1025, 2010.
    • (2010) Hypertension , vol.56 , pp. 1021-1025
    • Kurtz, T.W.1
  • 23
    • 77249147353 scopus 로고    scopus 로고
    • Recent advances in genetics of the spontaneously hypertensive rat
    • PRAVENEC M, KURTZ TW. Recent advances in genetics of the spontaneously hypertensive rat. Curr Hypertens Rep 12. 5-9, 2010.
    • (2010) Curr Hypertens Rep , vol.12 , pp. 5-9
    • Pravenec, M.1    Kurtz, T.W.2
  • 24
    • 0024517658 scopus 로고
    • An analysis of spontaneous hypertension in spontaneously hypertensive rats by means of new recombinant inbred strains
    • PRAVENEC M, KLÍR P, KŘEN V, ZICHA J, KUNEŠ J. An analysis of spontaneous hypertension in spontaneously hypertensive rats by means of new recombinant inbred strains. J Hypertens 7. 217-221, 1989.
    • (1989) J Hypertens , vol.7 , pp. 217-221
    • Pravenec, M.1    Klír, P.2    Křen, V.3    Zicha, J.4    Kuneš, J.5
  • 27
    • 0242413298 scopus 로고    scopus 로고
    • Transgenic and recombinant resistin impair skeletal muscle glucose metabolism in the spontaneously hypertensive rat
    • PRAVENEC M, KAZDOVÁ L, LANDA V, ZÍDEK V, MLEJNEK P, JANSA P, WANG J, QI N, KURTZ TW. Transgenic and recombinant resistin impair skeletal muscle glucose metabolism in the spontaneously hypertensive rat. J Biol Chem 278. 45209-45215, 2003.
    • (2003) J Biol Chem , vol.278 , pp. 45209-45215
    • Pravenec, M.1    Kazdová, L.2    Landa, V.3    Zídek, V.4    Mlejnek, P.5    Jansa, P.6    Wang, J.7    Qi, N.8    Kurtz, T.W.9
  • 39
    • 84877707375 scopus 로고    scopus 로고
    • One-step generation of mice carrying mutations in multiple genes by CRISPR/Cas-mediated genome engineering
    • WANG H, YANG H, SHIVALILA CS, DAWLATY MM, CHENG AW, ZHANG F, JAENISCH R. One-step generation of mice carrying mutations in multiple genes by CRISPR/Cas-mediated genome engineering. Cell 153. 910-918, 2013.
    • (2013) Cell , vol.153 , pp. 910-918
    • Wang, H.1    Yang, H.2    Shivalila, C.S.3    Dawlaty, M.M.4    Cheng, A.W.5    Zhang, F.6    Jaenisch, R.7


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.