Can the calcium hypothesis explain synaptic loss in alzheimer's disease?

Neurodegenerative Diseases

Volumn 13, Issue 2-3, 2014, Pages 139-141

  Popugaeva, Elena ^a   Bezprozvanny, Ilya ^a,b  

^a PETER THE GREAT ST PETERSBURG POLYTECHNIC UNIVERSITY   (Russian Federation)

^b UNIVERSITY OF TEXAS SOUTHWESTERN MEDICAL CENTER   (United States)

Author keywords

Alzheimer's disease; Calcium signaling; Mushroom spines; Neuronal store operated channels; Synapse

Indexed keywords

PRESENILIN; CALCIUM; INOSITOL TRISPHOSPHATE; RYANODINE RECEPTOR;

ALZHEIMER DISEASE; ARTICLE; CALCIUM HOMEOSTASIS; ENDOPLASMIC RETICULUM; GENE MUTATION; PATHOGENESIS; PRIORITY JOURNAL; SYNAPTIC TRANSMISSION; SYNAPTOSOME; AGING; AMNESIA; CONFERENCE PAPER; CRYSTAL STRUCTURE; DENDRITIC SPINE; DOWN REGULATION; NONHUMAN; SIGNAL TRANSDUCTION; SYNAPSE; UPREGULATION;

ALZHEIMER DISEASE; ANIMALS; CALCIUM; HUMANS; SYNAPSES;

EID: 84896829426     PISSN: 16602854     EISSN: 16602862     Source Type: Journal    
DOI: 10.1159/000354778     Document Type: Article

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