Alzheimer's disease and CADASIL are heritable, adult-onset dementias that both involve damaged small blood vessels

Cellular and Molecular Life Sciences

Volumn 71, Issue 6, 2014, Pages 949-955

  Marchesi, Vincent T ^a  

^a YALE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Alzheimer's dementia; Blood vessel damage; CADASIL; NOTCH 3

Indexed keywords

AMYLOID BETA PROTEIN; ANTIINFLAMMATORY AGENT; ANTIOXIDANT; NOTCH3 RECEPTOR;

AEROBIC METABOLISM; ALZHEIMER DISEASE; APOPTOSIS; ARTICLE; BLOOD BRAIN BARRIER; BRAIN BLOOD FLOW; BRAIN DAMAGE; BRAIN ISCHEMIA; CADASIL; DRUG TARGETING; DRUG TREATMENT FAILURE; HUMAN; INFLAMMATION; METABOLIC REGULATION; MICROANGIOPATHY; NEUROPATHOLOGY; NEUROTOXICITY; NEUROTRANSMISSION; NONHUMAN; OXIDATION;

ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN PRECURSOR; APOPTOSIS; BRAIN; CADASIL; CEREBRAL ARTERIES; HUMANS; INFLAMMATION; OXIDATIVE STRESS; PLAQUE, AMYLOID; PRESENILINS; RECEPTORS, NOTCH;

EID: 84896737366     PISSN: 1420682X     EISSN: 14209071     Source Type: Journal    
DOI: 10.1007/s00018-013-1542-7     Document Type: Article

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