Intermittent hypoxia-induced increases in reactive oxygen species activate NFATc3 increasing endothelin-1 vasoconstrictor reactivity

Vascular Pharmacology

Volumn 60, Issue 1, 2014, Pages 17-24

  Friedman, J K ^a   Nitta, C H ^a   Henderson, K M ^a   Codianni, S J ^a   Sanchez, L ^a   Ramiro Diaz, J M ^a   Howard, T A ^a   Giermakowska, W ^a   Kanagy, N L ^a   Gonzalez Bosc, L V ^a  

^a UNIVERSITY OF NEW MEXICO HEALTH SCIENCES CENTER   (United States)

Author keywords

Arteries; Hypertension; Intermittent hypoxia; Oxidative stress; Sleep apnea

Indexed keywords

CARBON DIOXIDE; ENDOTHELIN 1; MESSENGER RNA; REACTIVE OXYGEN METABOLITE; TEMPOL; TRANSCRIPTION FACTOR NFAT;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BLOOD PRESSURE; BLOOD VESSEL WALL; CONTROLLED STUDY; FEMALE; HYPERTENSION; IN VIVO STUDY; INTERMITTENT HYPOXIA; MALE; MESENTERIC ARTERY; MOUSE; NONHUMAN; PRIORITY JOURNAL; RAT; SLEEP DISORDERED BREATHING;

ARTERIES; HYPERTENSION; INTERMITTENT HYPOXIA; OXIDATIVE STRESS; SLEEP APNEA;

ANIMALS; ANOXIA; CALCIUM; ENDOTHELIN-1; FEMALE; KV1.5 POTASSIUM CHANNEL; MALE; MICE; MICE, INBRED BALB C; NFATC TRANSCRIPTION FACTORS; PROTEIN CARBONYLATION; RATS; REACTIVE OXYGEN SPECIES; SLEEP APNEA SYNDROMES; TRPC CATION CHANNELS; VASOCONSTRICTION;

EID: 84892507201     PISSN: 15371891     EISSN: 18793649     Source Type: Journal    
DOI: 10.1016/j.vph.2013.11.001     Document Type: Article

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