Salidroside improves doxorubicin-induced cardiac dysfunction by suppression of excessive oxidative stress and cardiomyocyte apoptosis

Journal of Cardiovascular Pharmacology

Volumn 62, Issue 6, 2013, Pages 512-523

  Wang, Xu Lei ^a   Wang, Xue ^b   Xiong, Li Li ^a   Zhu, Ye ^b   Chen, Hua Li ^b   Chen, Jia Xiang ^b   Wang, Xiao Xiao ^b   Li, Ru Li ^b   Guo, Zhi Yun ^a   Li, Ping ^a   Jiang, Wei ^b  

^a SOUTHWEST JIAOTONG UNIVERSITY   (China)

^b SICHUAN UNIVERSITY   (China)

Author keywords

Bcl2 Bax; Catalase; Doxorubicin; Mn SOD; ROS; Salidroside

Indexed keywords

CASPASE 3; CATALASE; DOXORUBICIN; LACTATE DEHYDROGENASE; MANGANESE SUPEROXIDE DISMUTASE; PROTEIN BAX; PROTEIN BCL 2; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE 1; SALIDROSIDE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ANTIOXIDANT ACTIVITY; APOPTOSIS; ARTICLE; BIOTECHNOLOGY; CARDIOTOXICITY; CELL DAMAGE; CELL SURVIVAL; CELL VIABILITY; CONTROLLED STUDY; DRUG EFFECT; DRUG EXPOSURE; DRUG MECHANISM; ENZYME ACTIVITY; HEART MUSCLE CELL; MALE; MOUSE; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN EXPRESSION; WEIGHT REDUCTION;

ANIMALS; ANTIBIOTICS, ANTINEOPLASTIC; ANTIOXIDANTS; APOPTOSIS; CARDIOTONIC AGENTS; CELL LINE; CELL SIZE; CELL SURVIVAL; CLONE CELLS; DOXORUBICIN; GENE EXPRESSION REGULATION, ENZYMOLOGIC; GLUCOSIDES; HEART VENTRICLES; MALE; MICE; MICE, INBRED C57BL; MYOCYTES, CARDIAC; OXIDATIVE STRESS; OXIDOREDUCTASES; PHENOLS; RANDOM ALLOCATION; RATS; VENTRICULAR DYSFUNCTION;

EID: 84890568380     PISSN: 01602446     EISSN: 15334023     Source Type: Journal    
DOI: 10.1097/FJC.0000000000000009     Document Type: Article

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