High glucose-induced oxidative stress promotes autophagy through mitochondrial damage in rat notochordal cells

International Orthopaedics

Volumn 37, Issue 12, 2013, Pages 2507-2514

  Park, Eun Young ^a   Park, Jong Beom ^a  

^a Uijeongbu St Mary's Hospital   (South Korea)

Author keywords

Autophagy; Diabetes; High glucose concentration; Intervertebral disc degeneration; Mitochondral damage; Oxidative stress; Reactive oxygen species

Indexed keywords

BECLIN 1; CATALASE; GLUCOSE; MANGANESE SUPEROXIDE DISMUTASE; REACTIVE OXYGEN METABOLITE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; AUTOPHAGY; CONTROLLED STUDY; DIABETES MELLITUS; DISORDERS OF MITOCHONDRIAL FUNCTIONS; MALE; MEMBRANE POTENTIAL; NONHUMAN; NUCLEUS PULPOSUS; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT;

ANIMALS; APOPTOSIS REGULATORY PROTEINS; AUTOPHAGY; CATALASE; CELLS, CULTURED; DOSE-RESPONSE RELATIONSHIP, DRUG; GLUCOSE; MITOCHONDRIAL DEGRADATION; MODELS, ANIMAL; NOTOCHORD; OXIDATIVE STRESS; RATS; REACTIVE OXYGEN SPECIES; SUPEROXIDE DISMUTASE; TIME FACTORS;

EID: 84890549211     PISSN: 03412695     EISSN: 14325195     Source Type: Journal    
DOI: 10.1007/s00264-013-2037-8     Document Type: Article

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