Transient acidosis during early reperfusion attenuates myocardium ischemia reperfusion injury VIA PI3K-AKT-enos signaling pathway

Oxidative Medicine and Cellular Longevity

Volumn , Issue , 2013, Pages

  Qiao, Xin ^a,b   Xu, Jinjin ^c   Yang, Qing Jun ^a   Du, Yun ^a   Lei, Shaoqing ^c   Liu, Zhi Hong ^d   Liu, Xinwei ^b   Liu, Huimin ^c  

^a Department of Anesthesiology Chongqing Zhoushan Hospital   (China)

^b THE FIRST AFFILIATED HOSPITAL OF CHONGQING MEDICAL UNIVERSITY   (China)

^c RENMIN HOSPITAL OF WUHAN UNIVERSITY   (China)

^d Department of Cardiac Surgery Sun Yat sen Cardiovascular Hospital   (China)

Author keywords

[No Author keywords available]

Indexed keywords

HEALTH CARE; MEDICINE;

CARDIOPROTECTION; ISCHEMIA-REPERFUSION INJURY; ISOLATED RAT HEART; MYOCARDIAL ISCHEMIA-REPERFUSION INJURIES; REPERFUSION; SIGNALING PATHWAYS;

CELL CULTURE;

ENDOTHELIAL NITRIC OXIDE SYNTHASE; PHOSPHATIDYLINOSITOL 3 KINASE; PROTEIN KINASE B; 8-EPI-PROSTAGLANDIN F2ALPHA; ISOPROSTANE DERIVATIVE; NITRIC OXIDE;

ACIDOSIS; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; ENZYME ACTIVATION; HEART INFARCTION SIZE; HEART LEFT VENTRICLE ENDDIASTOLIC PRESSURE; HEART MUSCLE REPERFUSION; HEART PROTECTION; INTRACELLULAR SIGNALING; ISOLATED HEART; MALE; NONHUMAN; PROTEIN EXPRESSION; RAT; REPERFUSION INJURY; ANIMAL; COMPLICATION; ENZYMOLOGY; HEART FUNCTION TEST; ISCHEMIC POSTCONDITIONING; METABOLISM; MYOCARDIAL ISCHEMIA REPERFUSION INJURY; PATHOLOGY; PATHOPHYSIOLOGY; PERFUSION; SIGNAL TRANSDUCTION; SPRAGUE DAWLEY RAT;

RATTUS;

ACIDOSIS; ANIMALS; ENZYME ACTIVATION; HEART FUNCTION TESTS; ISCHEMIC POSTCONDITIONING; ISOPROSTANES; MALE; MYOCARDIAL REPERFUSION INJURY; NITRIC OXIDE; NITRIC OXIDE SYNTHASE TYPE III; PERFUSION; PHOSPHATIDYLINOSITOL 3-KINASES; PROTO-ONCOGENE PROTEINS C-AKT; RATS; RATS, SPRAGUE-DAWLEY; SIGNAL TRANSDUCTION;

MLCS; MLOWN;

EID: 84888864059     PISSN: 19420900     EISSN: 19420994     Source Type: Journal    
DOI: 10.1155/2013/126083     Document Type: Article

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