Pretreatment with TCDD exacerbates liver injury from Concanavalin A: Critical role for NK cells

Toxicological Sciences

Volumn 136, Issue 1, 2013, Pages 72-85

  Fullerton, Aaron M ^a   Roth, Robert A ^a   Ganey, Patricia E ^a  

^a MICHIGAN STATE UNIVERSITY   (United States)

Author keywords

Autoimmune; Chemokines; Dioxin; Inflammation; Liver

Indexed keywords

2,3,7,8 TETRACHLORODIBENZO PARA DIOXIN; CD69 ANTIGEN; CHEMOKINE; CONCANAVALIN A; GAMMA INTERFERON;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ANTIOXIDANT ACTIVITY; ARTICLE; AUTOIMMUNE HEPATITIS; BLOOD LEVEL; DISEASE EXACERBATION; DOSE RESPONSE; HISTOPATHOLOGY; LIVER INJURY; LIVER TOXICITY; MACROPHAGE; MOUSE; NATURAL KILLER CELL; NEUTROPHIL; NONHUMAN; T CELL DEPLETION;

AUTOIMMUNE; CHEMOKINES; DIOXIN; INFLAMMATION; LIVER.;

ANIMALS; ANTIGENS, CD; ANTIGENS, CD1D; ANTIGENS, DIFFERENTIATION, T-LYMPHOCYTE; CHEMOKINES; CONCANAVALIN A; DOSE-RESPONSE RELATIONSHIP, DRUG; DRUG-INDUCED LIVER INJURY; HOMEODOMAIN PROTEINS; INFLAMMATION MEDIATORS; LECTINS, C-TYPE; LIVER; LYMPHOCYTE ACTIVATION; MACROPHAGES; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; NATURAL KILLER T-CELLS; NEUTROPHIL INFILTRATION; TETRACHLORODIBENZODIOXIN; TIME FACTORS;

EID: 84887951612     PISSN: 10966080     EISSN: 10960929     Source Type: Journal    
DOI: 10.1093/toxsci/kft174     Document Type: Article

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