T lymphocytes are direct, aryl hydrocarbon receptor (AhR)-dependent targets of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD): AhR expression in both CD4+ and CD8+ T cells is necessary for full suppression of a cytotoxic T lymphocyte response by TCDD

Toxicology and Applied Pharmacology

Volumn 185, Issue 2, 2002, Pages 146-152

  Kerkvliet, Nancy I ^a   Shepherd, David M ^a,b   Baecher Steppan, Linda ^a  

^a OREGON STATE UNIVERSITY   (United States)

^b UNIVERSITY OF MONTANA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

2,3,7,8 TETRACHLORODIBENZO PARA DIOXIN; AROMATIC HYDROCARBON RECEPTOR;

ANIMAL CELL; ANIMAL MODEL; ANIMAL TISSUE; CELLULAR IMMUNITY; CONTROLLED STUDY; CYTOTOXIC T LYMPHOCYTE; FEMALE; GRAFT VERSUS HOST REACTION; HOST CELL; LYMPHOCYTE FUNCTION; MAJOR HISTOCOMPATIBILITY COMPLEX; MOUSE; NONHUMAN; PROTEIN EXPRESSION; REVIEW; T LYMPHOCYTE SUBPOPULATION; WEIGHT REDUCTION;

ANIMALIA;

EID: 0036944759     PISSN: 0041008X     EISSN: None     Source Type: Journal    
DOI: 10.1006/taap.2002.9537     Document Type: Review

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