Olfm4 deletion enhances defense against Staphylococcus aureus in chronic granulomatous disease

Journal of Clinical Investigation

Volumn 123, Issue 9, 2013, Pages 3751-3755

  Liu, Wenli ^a   Yan, Ming ^b   Sugui, Janyce A ^c   Li, Hongzhen ^a   Xu, Chengfu ^b   Joo, Jungsoo ^b   Kwon Chung, Kyung J ^c   Coleman, William G ^b   Rodgers, Griffin P ^a  

^a NATIONAL HEART LUNG AND BLOOD INSTITUTE   (United States)

^b NATIONAL INSTITUTE OF DIABETES AND DIGESTIVE AND KIDNEY DISEASES   (United States)

^c NATIONAL INSTITUTE OF ALLERGY AND INFECTIOUS DISEASES   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CATHEPSIN G; CXCL2 CHEMOKINE; DIPEPTIDYL PEPTIDASE I; GRANULOCYTE COLONY STIMULATING FACTOR; GRANULOCYTE MACROPHAGE COLONY STIMULATING FACTOR; INTERLEUKIN 12P40; INTERLEUKIN 1BETA; INTERLEUKIN 6; LEUKOCYTE ELASTASE; OLFACTOMEDIN 4; PLASMA PROTEIN; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE 2; UNCLASSIFIED DRUG; CTSC PROTEIN, MOUSE; CYBB PROTEIN, MOUSE; CYTOKINE; GLYCOPROTEIN; MEMBRANE PROTEIN; OLFACTOMEDIN 4, MOUSE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; ASPERGILLUS FUMIGATUS; BACTERIAL CLEARANCE; BACTERIAL STRAIN; CHRONIC GRANULOMATOUS DISEASE; CONTROLLED STUDY; ENZYME ACTIVITY; FUNGAL STRAIN; GENE DELETION; HOST RESISTANCE; IN VIVO STUDY; INTRACELLULAR KILLING; LUNG ASPERGILLOSIS; LUNG INFECTION; MOUSE; NEUTROPHIL; NONHUMAN; PRIORITY JOURNAL; PROTEIN BLOOD LEVEL; SERUM; STAPHYLOCOCCUS AUREUS; STAPHYLOCOCCUS INFECTION; WILD TYPE; ANIMAL; ASPERGILLOSIS; BLOOD; C57BL MOUSE; COMPLICATION; DEFICIENCY; ENZYMOLOGY; GENETICS; IMMUNOLOGY; KNOCKOUT MOUSE; MALE; METABOLISM; MICROBIOLOGY;

ANIMALS; ASPERGILLOSIS; ASPERGILLUS FUMIGATUS; CATHEPSIN C; CYTOKINES; GENE DELETION; GLYCOPROTEINS; GRANULOMATOUS DISEASE, CHRONIC; MALE; MEMBRANE GLYCOPROTEINS; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; NADPH OXIDASE; NEUTROPHILS; STAPHYLOCOCCAL INFECTIONS; STAPHYLOCOCCUS AUREUS;

EID: 84883500763     PISSN: 00219738     EISSN: 15588238     Source Type: Journal    
DOI: 10.1172/JCI68453     Document Type: Article

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