Activation state-dependent interaction between Gαq subunits and the Fhit tumor suppressor

Cell Communication and Signaling

Volumn 11, Issue 1, 2013, Pages

  Zuo, Hao ^a   Chan, Grace P W ^a   Zhu, Jing ^a   Yeung, Wendy W S ^a   Chan, Anthony S L ^a   Ammer, Hermann ^b   Wong, Yung H ^a  

^a HONG KONG UNIVERSITY OF SCIENCE AND TECHNOLOGY   (Hong Kong)

^b UNIVERSITY OF MUNICH   (Germany)

Author keywords

Fhit; G protein; Phospholipase C ; Src; Tumor suppression

Indexed keywords

DINUCLEOSIDE PHOSPHATE; FRAGILE HISTIDINE TRIAD PROTEIN; G PROTEIN COUPLED RECEPTOR; GUANINE NUCLEOTIDE BINDING PROTEIN; I KAPPA B KINASE; MITOGEN ACTIVATED PROTEIN KINASE; PHOSPHOLIPASE C BETA; PROTEIN GAQ; PROTEIN TYROSINE KINASE; RAS PROTEIN; STAT3 PROTEIN; UNCLASSIFIED DRUG; ACID ANHYDRIDE HYDROLASE; CALCIUM; DIADENOSINE TRIPHOSPHATE; GUANINE NUCLEOTIDE BINDING PROTEIN ALPHA SUBUNIT; INOSITOL PHOSPHATE; SMALL INTERFERING RNA; TUMOR PROTEIN; TUMOR SUPPRESSOR PROTEIN;

ARTICLE; CANCER GROWTH; CANCER INHIBITION; CARCINOMA CELL; CELL PROLIFERATION; CHIMERA; COLON CARCINOMA; CONTROLLED STUDY; ENZYME PHOSPHORYLATION; FHIT GENE; GENE ACTIVATION; GENE INACTIVATION; GENE MUTATION; HUMAN; HUMAN CELL; IMMUNOPRECIPITATION; MALIGNANT NEOPLASTIC DISEASE; PRIORITY JOURNAL; PROTEIN ANALYSIS; PROTEIN BINDING; PROTEIN DOMAIN; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN HYDROLYSIS; PROTEIN INTERACTION; REGULATORY MECHANISM; SIGNAL TRANSDUCTION; TUMOR SUPPRESSOR GENE; CELL SURVIVAL; GENETICS; METABOLISM; MUTATION; PHOSPHORYLATION; TUMOR CELL LINE;

ACID ANHYDRIDE HYDROLASES; CALCIUM; CELL LINE, TUMOR; CELL SURVIVAL; DINUCLEOSIDE PHOSPHATES; GTP-BINDING PROTEIN ALPHA SUBUNITS, GQ-G11; HUMANS; INOSITOL PHOSPHATES; MUTATION; NEOPLASM PROTEINS; PHOSPHORYLATION; RNA, SMALL INTERFERING; SRC-FAMILY KINASES; TUMOR SUPPRESSOR PROTEINS;

EID: 84881421528     PISSN: None     EISSN: 1478811X     Source Type: Journal    
DOI: 10.1186/1478-811X-11-59     Document Type: Article

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