Fast motor axon loss in SMARD1 does not correspond to morphological and functional alterations of the NMJ

Neurobiology of Disease

Volumn 54, Issue , 2013, Pages 169-182

  Krieger, Frank ^a   Elflein, Nicole ^a   Ruiz, Rocio ^b   Guerra, Joana ^c   Serrano, Antonio L ^c   Asan, Esther ^a   Tabares, Lucia ^b   Jablonka, Sibylle ^a  

^a UNIVERSITY OF WÜRZBURG   (Germany)

^b UNIVERSITY OF SEVILLE   (Spain)

^c UNIVERSITAT POMPEU FABRA   (Spain)

Author keywords

Motor axon; Neuromuscular junction; Neurotransmission; Nmd2J mouse; SMARD1

Indexed keywords

ADENOSINE TRIPHOSPHATASE; HELICASE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL LOSS; CELL STRUCTURE; CONTROLLED STUDY; DENERVATION; DISEASE COURSE; GASTROCNEMIUS MUSCLE; LUMBAR SPINAL CORD; MOTOR NEURON DISEASE; MOUSE; MUSCLE CELL; NERVE ENDING; NERVE SPROUTING; NEUROMUSCULAR SYNAPSE; NEUROTRANSMISSION; NONHUMAN; PERIKARYON; PRIORITY JOURNAL; PROTEIN DEFICIENCY; REINNERVATION; SCIATIC NERVE; SPINAL CORD MOTONEURON; SPINAL MUSCULAR ATROPHY WITH RESPIRATORY DISTRESS TYPE 1; VENTRAL ROOT;

ANIMALS; AXONS; DISEASE MODELS, ANIMAL; DNA-BINDING PROTEINS; IMMUNOHISTOCHEMISTRY; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; MICROSCOPY, CONFOCAL; MOTOR NEURONS; MUSCLE, SKELETAL; NEUROMUSCULAR JUNCTION; SPINAL MUSCULAR ATROPHIES OF CHILDHOOD; TRANSCRIPTION FACTORS;

EID: 84876327036     PISSN: 09699961     EISSN: 1095953X     Source Type: Journal    
DOI: 10.1016/j.nbd.2012.12.010     Document Type: Article

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