LKB1-regulated adaptive mechanisms are essential for neuronal survival following mitochondrial dysfunction

Human Molecular Genetics

Volumn 22, Issue 5, 2013, Pages 952-962

  Nguyen, Marc Germain ^a,d   Khacho, Mireille ^a   Patten, David A ^a   Screaton, Robert A ^b,c   Park, David S ^a   Slack, Ruth S ^a  

^a Faculty of Medicine   (Canada)

^b UNIVERSITY OF OTTAWA   (Canada)

^c CHILDREN S HOSPITAL OF EASTERN ONTARIO   (Canada)

^d UNIVERSITÉ DU QUÉBEC À TROIS RIVIÈRES   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

APOPTOSIS INDUCING FACTOR; PROTEIN KINASE LKB1;

ADAPTATION; ALLELE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BIOGENESIS; CELL SURVIVAL; CONTROLLED STUDY; DEGENERATIVE DISEASE; DISORDERS OF MITOCHONDRIAL FUNCTIONS; ENERGY METABOLISM; GENE DELETION; GLYCOLYSIS; IN VIVO STUDY; MITOCHONDRIAL RESPIRATION; MOUSE; NEUROMODULATION; NONHUMAN; PRIORITY JOURNAL; PROTEIN FUNCTION; REGULATORY MECHANISM;

ANIMALS; APOPTOSIS; APOPTOSIS INDUCING FACTOR; CELL SURVIVAL; ENERGY METABOLISM; HUMANS; MICE; MITOCHONDRIA; MITOCHONDRIAL DISEASES; NEURODEGENERATIVE DISEASES; NEURONS; PROTEIN-SERINE-THREONINE KINASES;

EID: 84873443493     PISSN: 09646906     EISSN: 14602083     Source Type: Journal    
DOI: 10.1093/hmg/dds500     Document Type: Article

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