The response of NaV1.3 sodium channels to ramp stimuli: Multiple components and mechanisms

Journal of Neurophysiology

Volumn 109, Issue 2, 2013, Pages 306-314

  Estacion, Mark ^a,b   Waxman, Stephen G ^a,b  

^a YALE UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b VA CONNECTICUT HEALTHCARE SYSTEM   (United States)

Author keywords

Persistent current; Ramp current; Sodium channel; Voltage clamp; Window current

Indexed keywords

GLUTAMIC ACID; LYSINE; SODIUM CHANNEL; SODIUM CHANNEL NAV1.3;

AMINO ACID SUBSTITUTION; ARTICLE; CHANNEL GATING; CONTROLLED STUDY; ELECTRIC POTENTIAL; EVOKED RESPONSE; HUMAN; HUMAN CELL; NERVE CELL; NERVE CELL MEMBRANE STEADY POTENTIAL; NERVE FIBER TRANSECTION; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; RAMP STIMULUS; RECEPTOR UPREGULATION; SPINAL GANGLION; STIMULUS;

ACTION POTENTIALS; ANIMALS; GANGLIA, SPINAL; HEK293 CELLS; HUMANS; MUTATION, MISSENSE; NAV1.3 VOLTAGE-GATED SODIUM CHANNEL; NEURONS; PROTEIN ISOFORMS; RATS;

EID: 84872417471     PISSN: 00223077     EISSN: 15221598     Source Type: Journal    
DOI: 10.1152/jn.00438.2012     Document Type: Article

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